The polarographic method for the measurement of the oxygen tension in tissue is a beneficial addition to the group of the methods available for the study of the peripheral circulation.In this paper, the author analysed 1) the blood flow change in the muscle and the skin, 2) role of the arteriovenous anstomosis (AVA), in combination with the skin temperature measurement by thermocouple, the finger volume plethysmograph and N²⁴ clearance.The summary and conclusion of the experiment were as follows.1) The apparatus used is simular to what was described by Davies and Brink. The reliability, and the influence of the physico-chemical factors were retested, and it was concluded that this method could be available for the study of the peripheral circulation.2) By arterial occlusion, the skin PG and muscle PG descended and by oxygen inhalation they ascended. In this respect some consideration was made about the oxygen consumption of tissue, the oxygen diffusion and the blood circulation time.3) It was found that the skin PG showed the spontaneous fluctuation and moreover, every simultaneous record at two points of the body showed high correlation. It seemed the fluctuation might be in close relationship with the blood flow change caused by the nervous control probably in the vicinity of the arterioles.4) When the skin PG and temperature were recorded simultaneously, both curves often showed close simularity. It was suggested that the skin temperature change might in some occasion indicate the skin capillary blood flow change. Contrally, in the cases of poor correlation between them the explanation may be possible by increased blood flow through the AVA, where it is supposed that the oxygen can scarecely pass through the wall and that fluctuation differs from that of the arteriole.5) Simultaneous record of the skin PG and finger volume plethysmograph were carried out and in some occasion they showed correlation. This is a interesting finding to evaluate each method.6) The reactive hyperemia after release of arterial occlusion and caloric vestibular stimulation were studied and no definite tendency was seen polarographically.7) The spontaneous fluctuation was seen in the muscle PG which as in the case of the skin PG showed close simularity when recorded at every two points. And, the muscle PG had no close correlation to the skin PG.8) The reaction to cold, "Hunting" phomenon by Lewis, was studied with simultaneous record of skin PG and skin temperature during finger cooling. Both curves showed the huntung in much simular way. And, at various points of the hunting shown by temperature curve, oxygen inhalation lets the PG elevate variously. The interpretation is that the hunting depends not mainly upon the increase of blood flow of AVA, but upon the change of capillary blood flow.9) The changes of the muscle PG and the skin PG in indirect thermal application were compared with the reports based upon the conventional methods. By polargraphic method it may be possible to analyse the blood flow changes in the skin and the muscle separately.Also, the blood flow change of muscle and skin in Adrenaline administration were studied.

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As very complicated evil effects result from thoracoplasty, it is dangerous to make postoperative prognostication from ECGs alone. The present author has attempted to infer the possibility of circulation collapse after thoracoplasty, using as data for inference preoperative ECGs, the amount of hemorrhage, and results of examination of the autonomic nervous function.

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In this report the relations of postoperative changes in ECGs to adhesion and thickening of the surgically-attacked pleura, and to decrease of lung capacity were investigated periodically.METHODS Electrocardiographic examinations were done in the manner described in Report V. Postoperative pleural lesion was examined through X-ray pictures. Non-existence and very mild forms of pleural adhesion and thickening were taken as negative findings, while moderate and advanced forms as positive findings. Calculation of lung capacity was based on Ebina's vital capacity index, and decrease within (-) 50% of the standard vital capacity was regarded as negative, and that over (-) 50% as positive. The cases who were negative on the above-mentioned two points were recorded as having Pathologic Item (-); those who were positive on either of the two as having Pathologic Item (+₁); and those who were positive on the both points as having Pathologic Item (+₂).RETSULS AND CONCLUSIONS(1) Cases who preoperatively gave normal ECGs in a rest perisod and after exercise.i) In most cases of segmental resection rapid normalization of ECGs occurred together with improvement of pleural lesion and lung capacity in the 6th postoperative month.ii) In cases of lobar resection ECGs showed gradual improvement parallel to that of pleural lesion and lung capacity 6 months after operation.iii) In cases of total resection no significant improvement was noted in ECGs and clinical symptoms. Also with ECGs taken after exercise the same relationship was demonstrated to exist between ECGs and clinical symptoms.(2) The cases who before operation were electrocardiographically normal in a rest period, but not after exercise, and those who were abnormal in a rest period as well as after exercise continued to show the same electrocardiographic abnormalities even 6 months after operation in spite of improvement of pleural lesion and lung capacity. That is, in these cases no parallel relationship was established between improvement of clinical symptoms and that of ECGs. In a few cases of segmental resection, however, the parallel relationship was noted.

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Changes in the waves of ECGs after pulmonary resection were pursued from the 2nd postoperative month to the 12th, and comparison of postoperative ECGs with the preoperative was made. Electrocardiographic findings after pulmonary resection were rather different from those after thoracoplasty.

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We measured the maximum and the minimum blood pressure of 27 pairs of senescent twins and of 16 senescent siblings. We, however, did not come to the conclusion that the blood pressure of senescent persons is ruled by the hereditary factors.

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