We reported the case of a 83-years-old ambidextrous woman with somatoparaphrania, unilateral neglect, and anosognosia, which are examined usually due to the damage of non-dominant hemisphere, after left thalamic hemorrhage. Interestingly, she had supernumerary phantom limb which is rarely reported after a stroke. Her consciousness was clear, however there were severe hemiparesis and sensory loss in her right extremities. Her general cognition was nearly normal nonetheless very mild aphasia. In MRI, there was not any lesion except for left thalamus and internal capsula. The supernumerary phantom limb and somatopara phrania disappeared in about two months after the onset. On the other hand, anosognosia was remaining in six months after the onset. As the mechanism of these symptoms, the change of the lateralization of brain function, which was suggested from the background of the ambidexter. In other word, most of the right hemisphere function and a part of verbal function may be located at left hemisphere in this case. Single photon emission CT (SPECT) showed the wide range of hypoperfusion on the left cerebral hemisphere even though hemorrhage was localized to the thalamus. These hypoperfusion tended to improve three month after onset with the improvement of most of symptoms such as hemispatial neglect and somatopara phrenia. From this point, diaschisis after localized thalamic hemorrhage, which were caused by the discon nection of fibers that project to the temporal lobe and parietal lobe from the thalamus may have influence the decline of the cortical function.