Journal of Temporomandibular Joint

The factors associated with discontinuation of medical treatment and protraction of the treatment period in temporomandibular disorder cases: a preliminary study

Yoshioki HAMAMOTO, Kazuo YASUKAWA, Iku YAMAMORI, Hajime SHIBATA, Masak ...

2008 Volume 20 Issue 1 Pages 1-5
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.1

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The factors associated with discontinuation of medical treatment and protraction of the treatment period, which are undesirable end states of medical treatment in temporomandibular disorder cases, were examined preliminarily. Eighty patients, who were diagnosed with MRI and treated in Yamagata University Medical Hospital in 2003, were included in this study.
The medical treatment was interrupted for one-sided judgment of the patient in 29 cases (36%), whereas the treatment was finished by agreement of both the patient and dentist in 51 cases (64%) among 80 cases. The rate of cases whose condition did not improve by treatment and the rate of cases in which trismus was the chief complaint at the time of the first medical examination were larger in the discontinuation group than the other group.
Nineteen patients (24%) were treated for more than 6 months. The rate of cases in which the patient had a psychological abnormality was larger in the protracted period group than the other group.

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Effect of human brain activity on clenching with and without splint

Eri MAKIHARA, Shin-ichi MASUMI, Tatsuro TANAKA, Yasuhiro MORIMOTO, Ken ...

2008 Volume 20 Issue 1 Pages 6-10
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.6

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Although a stabilization splint is used for treating temporomandibular disorders or bruxism, the treatment mechanism is not yet clear. The purpose of this study was to evaluate human brain activity on clenching with and without a stabilization splint. Fifteen subjects were instructed to perform clenching three times for thirty seconds each time, with and without a stabilization splint. All brain activities during these tasks were imaged by using f-MRI and brain activity areas were evaluated. The areas where signal intensity increased on clenching without a splint were found to be in the left prefrontal cortex, left pars opercularis, left cingulate gyrus, bilateral primary motor cortex, bilateral primary somesthetic cortex, bilateral premotor cortex, and bilateral supplementary motor cortex. The signal intensity in the primary motor cortex became in equilibrium by wearing a splint. The activities in the premotor cortex and supplementary motor cortex increased by wearing a splint.
These results suggested that the stabilization splint decreases abnormal masticatory muscle activity.

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Basic behavior of finite helical axis parameters during mastication

Kazuo HAYASHI, Itaru MIZOGUCHI

2008 Volume 20 Issue 1 Pages 11-15
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.11

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The purpose of this study was to define new parameters of finite helical axis (FHA) analysis for mandibular kinematics, especially mastication. One subject without signs and symptoms of temporomandibular disorders and with normal occlusion was selected. The chewing movement of the mouth was recorded by an opto-electric system (TRIMET system). In this study, the direction vector and position vector of the FHA, the rotation angle of the FHA and the translation along the FHA were used as the FHA parameters. One of the newly defined FHA parameters, the FHA direction vector, was useful to quantify the lateral movement of the mandible during chewing movement. Furthermore, the FHA position vectors were effective to clarify the vertical movement of the condyle during mastication. The FHA parameters developed in this study provide detailed information of mandibular kinematics.

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Disturbance at mouth closing due to posterior band of reduced articular disc associated with internal derangement of TMJ: a case report

Yuji TAKAYAMA, Ritsuo TAKAGI, Hisao AJIMA, Masaki NAGATA

2008 Volume 20 Issue 1 Pages 16-19
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.16

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The patient was a 35-year-old female who was referred to our clinic with the complaint of disturbance of mouth closing on November 9, 1998. She occasionally felt clicking sounds at her left temporomandibular joint when she opened and closed her mouth. At the end of October 1998, after opening her mouth widely, she was not able to completely close her mouth. MR images revealed displacement of the articular disc to the medial side at mouth closing, and that it was repositioned to its original position on the condylar head at mouth opening. Furthermore, the repositioned disc was often not able to displace again to the medial side of the condylar head, and interfered with mouth closing.
This symptom gradually worsened over 5 years, and so surgical removal of the articular disc was performed. She is now able to open and close her mouth without any trouble, postoperatively.

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Changes in extracellular matrix and MMP-3 expression in the mandibular condylar cartilage of collagen-induced arthritis rats

Takashi FUJII, Hirohito KUBO, Kenji KAKUDO

2008 Volume 20 Issue 1 Pages 20-26
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.20

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Rheumatoid arthritis (RA) is an autoimmune disease characterized by chronic synovitis and progressive destruction of the joints. In this study, we investigated changes in the extracellular matrix of mandibular condylar cartilage after experimentally inducing arthritis, using histological examination, immunohistochemistry and molecular biology.
According to changes in the gross score for the left hind paw in DA rats after immunization with type II collagen, all rats were evaluated with a score of over 3 after immunization at 18 days. Although mandibular condylar cartilage destruction was slight histologically, stainability for type II collagen, chondroitin 4-sulfate and chondroitin 6-sulfate gradually became weaker. The expression of MMP-3mRNA in the condylar cartilage showed an increase on quantitative real-time PCR 2 weeks after immunization. These findings suggest that changes in the extracellular matrix of the mandibular condylar cartilage occur in the early stage of arthritis and that MMP-3 plays a crucial role in the pathogenesis of arthritic destruction.

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[title in Japanese]

[in Japanese]

2008 Volume 20 Issue 1 Pages 28
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.28

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Musculoskeletal biomechanics: the temporomandibular joint

Christopher C. Peck

2008 Volume 20 Issue 1 Pages 29
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.29

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[title in Japanese]

[in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...

2008 Volume 20 Issue 1 Pages 30-31
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.30

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[title in Japanese]

[in Japanese], [in Japanese], [in Japanese], [in Japanese]

2008 Volume 20 Issue 1 Pages 32-33
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.32

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[title in Japanese]

[in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...

2008 Volume 20 Issue 1 Pages 34-35
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.34

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[title in Japanese]

2008 Volume 20 Issue 1 Pages 36-50
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.36

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[title in Japanese]

2008 Volume 20 Issue 1 Pages 51-57
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.51

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[title in Japanese]

2008 Volume 20 Issue 1 Pages 58-89
Published: April 20, 2008
Released on J-STAGE: June 28, 2010

DOIhttps://doi.org/10.11246/gakukansetsu1989.20.58

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