Idiopathic portal hypertension is clinically characterized by the elevated portal pressure with marked splenomegaly, anemia, no evidence of extrahepatic obstruction of the portal vein and absence of cirrhosis. Etiology and pathogenesis of the disease still remains obscure. In the present study, pathologic physiology of idiopathic portal hypertension was investigated with special emphasis on hemodynamic aspects including splenic, portal and hepatic arterial circulation.
In this paper splenic anemia is defined as a synonym of Banti's syndrome whicht is initiated by splenomegaly and chronic anemia, later followed by liver cirrhosis.