JAPANESE CIRCULATION JOURNAL

JAPANESE CIRCULATION JOURNAL

Online ISSN : 1347-4839
Print ISSN : 0047-1828
ISSN-L : 0047-1828

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Volume 28, Issue 1

Displaying 1-14 of 14 articles from this issue

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Etiologic Factor of Portal Hypertension in Chronic Hepatitis : Symposium on Methods for the Study of Hepatic Circulation; Hepatic Circulatory Disturbances in Liver Diseases

YOSHIHIRO SHIMADA

1964 Volume 28 Issue 1 Pages 1-7
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.1

JOURNAL FREE ACCESS

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^<198>Au Colloid Clearance and Liver Cell Clearance in Hepatic Injuries : Symposium on Methods for the Study of Hepatic Circulation; Hepatic Circulatory Disturbances in Liver Diseases

GYOICHI WAKISAKA, TERUO FUJITA

1964 Volume 28 Issue 1 Pages 7-10
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.7

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Hepatic Circulation in Liver Diseases : MEASUREMENT OF HEPATIC BLOOD FLOW IN SOME LIVER DISEASES AND ANALYSIS OF SYSTEMIC CIRCULATORY CHANGES IN LIVER CIRRHOSIS : Symposium on Methods for the Study of Hepatic Circulation; Hepatic Circulatory Disturbances in Liver Diseases

HARUO KAMEDA, MEISEI OHTA

1964 Volume 28 Issue 1 Pages 10-13
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.10

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Measurement of Blood Flow in Intra- and Extra-hepatic Shunt : Symposium on Methods for the Study of Hepatic Circulation; Hepatic Circulatory Disturbances in Liver Diseases

SHOZO NAKAMURA

1964 Volume 28 Issue 1 Pages 13-17
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.13

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Using radioisotope clearance method and dilution technique, various circulatory problems in liver diseases, chiefly in liver cirrhosis, were studied. 1.) Remarkable findings in circulatory changes of liver cirrhosis were not only de-creased liver blood flow but also increased cardiac output and peripheral blood flow. 2.) Method for separate determination of hepatic arterial and portal blood flow was further studied. 3.) The new method detecting the presence and degree of intrahepatic shunt was introduced .

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Hemodynamic Analysis on the Liver in Portal Hypertension : Symposium on Methods for the Study of Hepatic Circulation; Hepatic Circulatory Disturbances in Liver Diseases

YASUO KUROYANAGI, YASUHISA YOKOYAMA

1964 Volume 28 Issue 1 Pages 17-19
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.17

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Experimental Studies on the Clearance of Indocyanine Green from the Blood : Symposium on Methods for the Study of Hepatic Circulation; Hepatic Circulatory Disturbances in Liver Diseases

SADAKICHI HIROSE, HIRONAKA KAWASAKI, CHISATO HIRAYAMA, TOMIICHI MASUYA

1964 Volume 28 Issue 1 Pages 19-21
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.19

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Clinical investigations were carried out in 290 patients with portal hypertension. The patients were divided into 4 groups according to the site of the obstruction to the portal and hepatic veins, whether it is extra-or intra-hepatic. Mechanisms on ascites formation and Splenomegaly were discussed.

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Microscopic Observation of the Intralobular Blood Circulation and the Pressure Gradient of Hepatic Vascular Pathway : Symposium on Intrahepatic Microcirculation: Methods for the Study of Intra- or Extra-lobular Circulation; Hemodynamic and Morphological Aspects

KATSUJI NAKATA

1964 Volume 28 Issue 1 Pages 24-27
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.24

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The Sinusoidal Blood Flow and Pressure in the Liver Cirrhosis of the Rat : Symposium on Intrahepatic Microcirculation: Methods for the Study of Intra- or Extra-lobular Circulation; Hemodynamic and Morphological Aspects

SHIGEKI OKA, TATSUJI KANETAKA

1964 Volume 28 Issue 1 Pages 27-30
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.27

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Hemodynamic in Intra-hepatic Circulation and It's Signification on Behavior of Hepatic Artery in the Damaged Liver : Symposium on Intrahepatic Microcirculation: Methods for the Study of Intra- or Extra-lobular Circulation; Hemodynamic and Morphological Aspects

SHUJI HOSOKAWA

1964 Volume 28 Issue 1 Pages 30-33
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.30

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Studies on Intrahepatic Vascular Alterations in Cirrhosis of the Liver : Symposium on Intrahepatic Microcirculation: Methods for the Study of Intra- or Extra-lobular Circulation; Hemodynamic and Morphological Aspects

MASASHI MIYAKE, MAMORU SAITO, MASAHIKO OKUDAIRA

1964 Volume 28 Issue 1 Pages 33-39
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.33

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The Histophysiological Investigation of the Lymphatic System in the Liver : Symposium on Intrahepatic Microcirculation: Methods for the Study of Intra- or Extra-lobular Circulation; Hemodynamic and Morphological Aspects

SUMIKO MAGARI

1964 Volume 28 Issue 1 Pages 39-41
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.39

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The authors reported several characteristic patterns of the intrahepatic vascular alterations in the cirrhosis and the other pathological conditions of the liver, in association with a consideration of hemodynamic points of view.

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Ascites Formation and Changes of Microcirculation in the Liver : Symposium on Intrahepatic Microcirculation: Methods for the Study of Intra- or Extra-lobular Circulation; Hemodynamic and Morphological Aspects

EIICHI KATO, MASAHARU TSUCHIYA

1964 Volume 28 Issue 1 Pages 41-45
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.41

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On the basis of these experimental findings, the following sequence of events can be postulated as the mechanism of ascites formation : increased permeability of sinusoids resulting from disturbance of postsinusoidal out-flow leads to an increase in production of the lymphatic fluid, to the extent that the capability of lymphatic drainge from the liver into the thoracic duct is exceeded and part of the lymphatic fluid is regurgitated through the capsule of the liver into the peritoneal cavity. The increase of thoracic duct pressure also causes congestion of lymphatic vessels and of small veins in the mesenterium peritoneum and intestinal wall which result in increased extravasation and decreased resorption of the ascitic fluid. This sequence of events, in conjunction with other factors as mentioned in the beginning of this article, contributes to establishment of a viscious cycle which makes ascites sometimes very resistant to treatment. It seems that the supression of sinusoid permeability and the acceleration of a drainage of hepatic lymph into the vein are the most important and essential in the treatment of the hepatogenous ascites formation.

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EFFECT OF PREGNANCY ON THE COURSE OF EXPERIMENTALLY AND SPONTANEOUSLY HYPERTENSIVE RATS : Experimental Study on the Blood Pressure of Pregnant Hypertensive Rats

TOSHIO TAKEDA

1964 Volume 28 Issue 1 Pages 49-54
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.49

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1. A study has been made of the effect of pregnancy on the level of the blood pressure in 86 pregnancies of 10 normotensive, 54 experimentally hypertensive, and 7 spontaneously hypertensive rats of Wistar strains. 2. No appearance of eclampsia-like syndrome or increased tendency of abortion or fetal death was observed in those pregnant rats with experimental or spontaneous hypertension. 3 . High blood pressure dropped moderately at late pregnancy in all kinds of experimental hypertension (renal infarction, renal ablation, adrenal regeneration, DCA and Post DCA, and salt hypertension) and in the spontaneous hypertension investigated in this study. The normotensive rats did not show any definite change during pregnancy. 4. In renal infarction and adrenal regeneration hypertension, the degree of blood pressure fall at late pregnancy in acute hypertensives was more marked than that in long continued hypertensives. 5. The degree of blood pressure fall correlated significantly with the litter weight or the number of young in rats with renal infarction hypertension.

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THE MECHANISM OF THE FALL OF BLOOD PRESSURE AT LATE PREGNANCY IN EXPERIMENTALLY HYPERTENSIVE RATS : Experimental Study on the Blood Pressure of Pregnant Hypertensive Rats

TOSHIO TAKEDA

1964 Volume 28 Issue 1 Pages 55-63
Published: January 20, 1964
Released on J-STAGE: April 14, 2008

DOIhttps://doi.org/10.1253/jcj.28.55

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The mechanism of the fall of blood pressure during pregnancy in experimentally hypertensive rats was studied in this report. 1. Fetal ablation carried out on the 16th day of pregnancy without damage to the placenta interfered with the fall of blood pressure in hypertensive rats with renal infarction. 2. Intraperitoneal transplantation of new born kidney, liver, spleen or placenta as cell suspension or parenteral injection of placental homogenate to renal hypertensive rats was per-formed and the changes of blood pressure by these procedures were studied. In these studies, only new born kidney transplantation led to a definite fall of blood pressure. Histologically the viable epithelial cells of new born kidney assumed a tubular structure or irregular cluster in the peritoneal granule or lymphnode. 3. Judging from these data, the fetus kidneys may play an important role in the fall of blood pressure at late pregnancy in experimentally and spontaneously hypertensive rats, that is, the fetus kidney may secrete some antihypertensive substances which can lower the blood pressure of pregnant hypertensive rat.

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