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Volume 8, Issue 2
Displaying 1-5 of 5 articles from this issue
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Akira OHARA, Kimiko TODA1967 Volume 8 Issue 2 Pages 45-52
Published: 1967
Released on J-STAGE: August 29, 2006
JOURNAL FREE ACCESSRadiolysis of tyrosin, phenylalanine and 3, 4-dihydroxyphenylalanine in aqueous solution has been studied quantitatively. It was found that γ-irradiation caused on (oxidative) deamination, yielding ammonia and the corresponding a-keto acids. Comparison of the rate of radiolytic decomposition has shown that of the three amino acid, phenylanine is most sensitive and tyrosin is most resistant to γ-radiation.View full abstractDownload PDF (276K) -
Akira TSUYA, Shigeki OKANO1967 Volume 8 Issue 2 Pages 53-64
Published: 1967
Released on J-STAGE: August 29, 2006
JOURNAL FREE ACCESSThe relative biological effectiveness of neutron radiation of 0.43, 0.65, 1.0, 1.5, 1.8 and 14.1 Mev was studied, taking the dosis for halving the labeling index of 3H-thymidine as an index.
Adult female mice of ddN strain and Carworth Farms BNL Stoner strain were exposed to single whole-body radiation of 250 kvp X-ray, 60Co gamma-ray and neutron radiation.
1) The RBE of total bone marrow cell and red cell precursor for 0.43 Mev neutron is highest, 4.1 and 3.6 respectively, with decreasing RBE for lowering LET. The RBE of total bone marrow cell is 1.2 for 14.1 Mev fast neutron.
It is considered that there exists maximum RBE of neutron in the low energy range of less than 2 Mev.
2) The RBE of 14.1 Mev fast neutron are 1.1 and 1.2 for thymus and spleen, respectively.
3) The RBE of 14.1 Mev fast neutron is 1.8 for the intestine. The higher RBE of the intestine suggest higher radiosensitivity of this organ than others.View full abstractDownload PDF (390K) -
Nagasuini YAGO, Shigeru KOBAYASHI, Saburo OMATA, Shogo ICHII1967 Volume 8 Issue 2 Pages 65-71
Published: 1967
Released on J-STAGE: August 29, 2006
JOURNAL FREE ACCESSThe effect of lethal dose (1000 R) of whole-body X-irradiation on levels of α-glucan phosphorylase and glycogen in livers of intact and adrenalectomized rats was studied. Both total and resting activities of α-glucan phosphorylase in livers of irradiated, fasted rats remained at the level of unirradiated, ad libitum fed rats for a total period of 72 hours post-irradiation. This was in contrast with the progressive decrease in the activity of this enzyme in unirradiated, fasted rats. Whole-body X-irradiation of adrenalectomized rats did not appear to afford any such effect of maintaining the level of α-glucan phosphorylase higher than unirradiated, fasted animals. The relationship between levels of α-glucan phosphorylase and glycogen in livers of irradiated rats was discussed.View full abstractDownload PDF (296K) -
Masami KIGA, Isao TAZAKI1967 Volume 8 Issue 2 Pages 72-79
Published: 1967
Released on J-STAGE: August 29, 2006
JOURNAL FREE ACCESSAn analysis was carried out to explain the shape of survival curves in fractionated irradiation. The dose which seemed to remain in the irradiated animals at the time of subsequent exposure was estimated from (differential) survival decrease by the exposure. Single-dose survival curve of the experimental animals was the guide to the estimation.
The apparently remained dose in animals surviving previous dose at the time of each successive exposure assumed to be “residual dose” or RD, which was denoted from residual damage to which not differential but total survival decrease was referred.
Recovery rate was calculated from our experiment of whole body irradiation in mice. Dose consisted in residual dose at the time of i-th exposure together with the i-th exposure decreasing during the timeinterval to the residual dose at the time of the subsequent i + 1—th exposure, indicated the recovery rate being independent to dose size. However, the recovery did not seem exponential to interval days ; two days recovery seemed most rapid.View full abstractDownload PDF (234K) -
Akiko UENO, Yasukazu AKITA1967 Volume 8 Issue 2 Pages 80-91
Published: 1967
Released on J-STAGE: August 29, 2006
JOURNAL FREE ACCESSEffect of X-irradiation on myosin-ATPase has been studied. It was found that on X-irradiation, ATPase activity of myosin was markedly increased, if a certain amount of histidine was present in the solution. It was also found that when an aqueous solution of histidine was first irradiated with X-rays and then this irradiated histidine added to a myosin solution, an equally marked activation of myosin ATPase was produced. These results seem to indicate that some kinds of activating agent for myosin-ATPase was formed from histidine on irradiation.
Irradiated histamine and imidazol were also found to be effective in producing a marked activation of ATPase when these were added to myosin solutions. However, when myosin solutions that contained either histamine or imidazol were irradiated, ATPase activity of these solutions was not so high as that observed when a myosin solution, containing histidine, were irradiated.
Possible mechanisms involved in the activation of myosin-ATPase by X-irradiation in the presence of histidine have been discussed.View full abstractDownload PDF (394K)
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