魚病研究
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43 巻 , 2 号
選択された号の論文の6件中1~6を表示しています
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論文
  • Carmelo Segovia del Castillo, Takeshi Yoshikawa, Masahito Hashimoto, T ...
    43 巻 (2008) 2 号 p. 65-71
    公開日: 2008/07/08
    ジャーナル フリー
    The anti-bacterial substance, AVS-03d, isolated from the culture of marine Pseudoalteromonas sp. A1-J11 was separated by HPLC with a C18 column. The chemical structure of AVS-03d was identified as 2-n-pentyl-4-quinolinol (C14H17NO = 215.13) on the basis of mass spectrometry and NMR spectroscopy. AVS-03d showed strong inhibitory activity against Vibrio harveyi strains as compared with other quinolinol compounds, suggesting that the length of the alkyl side chain of the compound is important for anti-bacterial activity.
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  • Yoshisuke Iida, Junya Hiroi, Kenji Namba, Toshihiro Nakai
    43 巻 (2008) 2 号 p. 72-78
    公開日: 2008/07/08
    ジャーナル フリー
    Flounder herpesvirus (FHV) induces epidermal hyperplasia in larval Japanese flounder Paralichthys olivaceus. We examined the influence of ambient oxygen and salinity concentrations on mortality of flounder larvae by FHV infection. FHV-infected fish showed markedly higher mortality than uninfected fish under normoxia (partial oxygen pressure; PO2 = ca. 160 Torr), but survived at high rates under hyperoxia (PO2 = ca. 280 Torr or higher) as in uninfected fish. In the PO2 range from 100 to 400 Torr, infected fish always displayed lower levels of oxygen consumption (MO2) compared to the uninfected fish. Under PO2 of 260 Torr, the 48 h-survival rate of infected fish in diluted seawater (salinity 8 or 16 ppt) was much higher than that in full-strength seawater (salinity 32 ppt). Whole-mount immunocytochemistry to detect Na+/K+-ATPase and Na+/K+/2Cl- cotransporter of the skin of both infected and uninfected larvae revealed that the infected larvae had a significantly lower number of chloride cells. These results suggest that flounder larvae infected with FHV die of dysfunction in respiration and osmotic regulation.
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  • 田中 眞, 佐藤 孝幸, 馬 文君, 小野 信一
    43 巻 (2008) 2 号 p. 79-82
    公開日: 2008/07/08
    ジャーナル フリー
    Viral endothelial cell necrosis of eel (VECNE) of Japanese eel Anguilla japonica, caused by an adenovirus (JEAdV), has been a serious problem of aquaculture industry in Japan. In the present study, effectiveness of increasing water temperature and non-feeding against VECNE was evaluated. Cumulative mortalities of fish intraperitoneally injected with 105.05 TCID50/fish of JEAdV increased with elevating water temperature in the range between 20°C and 31°C, but mortality at 35°C was as low as that at 20°C. Rearing infected fish under the non-feeding condition further enhanced the effect of treatment at 35°C. The effectiveness was dependent on rearing periods at the high temperature; more than 3 days at 35°C were needed to reduce mortality. Fish, which survived the primary challenge with JEAdV at 35°C, showed high resistance to re-challenge with JEAdV. From these results, the treatment of fish under the non-feeding condition at 35°C is useful to control VECNE.
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