Advances in Animal Cardiology
Online ISSN : 1883-5260
Print ISSN : 0910-6537
ISSN-L : 0910-6537
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Volume 31, Issue 2
Displaying 1-5 of 5 articles from this issue
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  • Hemodynamics of Angiotensin Converting Enzyme Inhibitors
    Masami UECHI
    1998 Volume 31 Issue 2 Pages 53-58
    Published: 1998
    Released on J-STAGE: September 17, 2009
    DOIhttps://doi.org/10.11276/jsvc1984.31.53
    JOURNAL FREE ACCESS
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    Several large, prospective, randomized, placebo-controlled trials have demonstrated that treatment with Angiotensin Converting Enzyme (ACE) inhibitors results in a reduction in overall mortality in human and small animal, patients with heart failure. ACE regulates the balance between the vasoconstrictive, salt-retensive, and hypertrophic properties of angiotensin II and the vasodilatory and naturetic properties of bradykinin. ACE inhibitors alter this balance by decreasing the formation of angiotensin II and the degradation of bradykinin. ACE inhibitors also indirectly alter the metabolism of a number of other vasoactive substances and diuretic hormon; NO, prostagrandin, and endothelin, and ADH, etc. ACE inhibitors decrease systemic vascular resistance but cause little change in heart rate. ACE inhibitors have effect on cardiac output or pulmonary capillary wedge pressure. It is clear that ACE inhibitors represent one of major advances in cardiovascular therapeutics in small animal cardiovascular medicine.
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  • The Current Therapy of Chronic Heart Failure by Angiotensin Converting Enzyme Inhibitor
    Naoyuki TAKEMURA
    1998 Volume 31 Issue 2 Pages 59-68
    Published: 1998
    Released on J-STAGE: September 17, 2009
    DOIhttps://doi.org/10.11276/jsvc1984.31.59
    JOURNAL FREE ACCESS
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    In the present commentary, the clinical use and the associated side effects of angiotensin converting enzyme inhibitors (ACEIs), the cornerstone in the long-term treatment of chronic heart failure (CHF) in the small animal practice, were reviewed. Based on the present author's unpublished data, it was indicated that ACEIs might suppress the progression of CHF when they could be given to a dog with mild hear failure. Incresed blood urea nitrogen (BUN) during chronic angiotensin converting enzyme inhibition was also discussed.
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  • Left Hepatic Venous Connection to Left Atrium in a Calf
    Takayuki MURAKAMI, Kazuyuki UCHIDA, Kouji OHWADA, Syunichi KAIZOU
    1998 Volume 31 Issue 2 Pages 69-73
    Published: 1998
    Released on J-STAGE: September 17, 2009
    DOIhttps://doi.org/10.11276/jsvc1984.31.69
    JOURNAL FREE ACCESS
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    Left hepatic vein opening to the left atrium was observed in a 36-day-old Japanese black calf. Two middle hepatic and a right hepatic veins opened normally to the posterior vena cava. The heart was associated with absence of the coronary sinus, anomalous opening of the great cardiac vein to the left atrium, atrial septal defect, large two ventricular septal defects and straddling right atrioventricular valve. The embryonic development of the left hepatic venous connection to the left atrium was discussed.
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  • Fibrous Endocarditis in a Reticulated Giraffe (Giraffa camelopardalis reticulata)
    Yoshihisa SEKI, Yoko KASHIDA, Noboru MACHIDA, Keiji KIRYU
    1998 Volume 31 Issue 2 Pages 74-77
    Published: 1998
    Released on J-STAGE: September 17, 2009
    DOIhttps://doi.org/10.11276/jsvc1984.31.74
    JOURNAL FREE ACCESS
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    An 18-year-old female reticulated giraffe (Giraffa camelopardalis reticulata), which had shown signs of congestive heart failure, was examined cardiopathologically. The heart was grossly enlarged and rounded. Both ventricular cavities were noticeably dilated. In the left ventricular wall, two hard masses of greyish-whitish in color were found on the upper portions of the anterior and posterior papillary muscles (6×4cm and 8×3cm, respectively). The chordae tendineae and a part of the leaflets of the mitral valve were involved in both of the mass lesions. On the endocardial surface of the left atrium, there were opaque, wrinkled areas of endocardial thickening characteristic of jet lesions. Microscopically, the mass lesions were of organized thrombotic endocarditis in the chordae tendineae, regarded as fibrous endocarditis.
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  • A Feline Case of Dilated Cardiomyopathy Observed Clinically for a Long Time
    Hiroshi KOIE, Naofumi OGUSHI, Masato KITAGAWA, Takao KANAI, Shigeo TAN ...
    1998 Volume 31 Issue 2 Pages 78-83
    Published: 1998
    Released on J-STAGE: September 17, 2009
    DOIhttps://doi.org/10.11276/jsvc1984.31.78
    JOURNAL FREE ACCESS
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    A long-term observation of a feline case of dilated cardiomyopathy was performed by echocardiography. The left ventricular wall was gradually dilated. The cardiac wall thickness measured by echocardiography was approximately consistent with necropsy result. It was suggested that echocardiography was useful for observation of changing wall thickness.
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