Kanzo
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Print ISSN : 0451-4203
ISSN-L : 0451-4203
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Volume 21, Issue 10
Displaying 1-34 of 34 articles from this issue
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  • Pathogenesis of vacuolar degeneration of the hepatocyte
    Significance of intrasinusoidal pressure elevation
    Yuro SHIBAYAMA, Kazuki MATSUMOTO, Masafumi SAITOH, Katsuji NAKATA
    1980 Volume 21 Issue 10 Pages 1283-1294
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1283
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    It is generally believed that the hepatocytes are extremely vulnerable to hypoxia, and that vacuolar degeneration of the hepatocyte is the earliest light microscopic change in hypoxia. To analyse the pathogenesis of the vacuolar degeneration, the present study was carried out by isolated liver perfusion experiments. In severe hypoxia, a decrease in hepatic function was recognized, but no vacuolar degeneration developed at all. In hyperoxia, vacuolar degeneration appeared in the hepatocytes of the central zone at a inferior vena cava pressure of 60-70mm H2O, and also developed in the hepatocytes of the peripheral zone with the higher pressure. Moreover, vacuolar degeneration appeared in the hepatocytes of the peripheral zone at a portal vein pressure of 160mm H2O. It was concluded that the development of vacuolar degeneration of the hepatocyte is closely related with the increase in the intrasinsoidal pressure and not with hypoxia. As for the reason why the vacuolar degeneration generally appear in the hepatocytes of the central zone, we would suggest the hypothesis that the critical sinusoidal pressure for vacuole formation of the hepatocytes of the central zone is lower than that of the hepatocytes of the peripheral zone.
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  • Clinical studies on an outbreak of hepatitis A in a junior high school
    2. Clinical and laboratory course
    Masami IMOTO, Satoshi NAKANO, Hajime WATAHIKI, Kimio KITAMURA, Isao TA ...
    1980 Volume 21 Issue 10 Pages 1295-1303
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1295
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    The outbreak of hepatitis A occurred in a junior high school of Ghodo town, located in the rural district of Ogaki city. The clinical course and time change of abnormal laboratory data of fifty patients who admitted to Ogaki municipal hospital and two other hospitals in the town were reported.
    Fever, nausea and general fatigue disappeared within a week in almost all patients. Hepatomegaly continued over three months in 25%. The duration of abnormal S-GOT, S-GPT and IgM was longest in icteric group.
    The laboratory recovery needed more days in the group, which γ-GTP was high at onset of the illness. Recovery of total bilirubin, S-GPT and IgM was delayed in the group which TTT elevated for many months or re-elevated. TTT seems to be a good indicator for prolongation of this illness.
    The time change of anti HA titer showed biphasic curve and the two peaks were observed at 2 week and 6 month after onset of the illness. Initial peak coincidates with that of IgM.
    Reelevation of transaminase and gradual elevation of total bilirubin were observed in 4 and 2 cases respectively.
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  • The detection of e-antigen and e-antibody by the radioimmunoassay (Dainabot Co., Ltd.) was investigated and it was compared with the immunodiffusion (ID) method
    Hiroshi SUZUKI, Shiro IINO, Fumihiro ICHIDA, Shiro SUZUKI, Akira YOSHI ...
    1980 Volume 21 Issue 10 Pages 1304-1309
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1304
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    1) By RIA, 86% of the tested cases are positive of either e-antigen or anti-e, and only 5% of them are both negative. The cases with the judge pended are 9% of them.
    2) Although the co-existence of e-antigen and e-antibody was obviously confirmed only in 3 out of 774 cases, there were some cases among the cases with judge pended, where both e-antigen and e-antibody were considered to co-exiSt with each other.
    3) The number of cases negative for both e-antigen and e-antibody and of cases with judge pended was the smallest among the asymptomatic carriers, followed by carriers with chronic hepatitis, and by carriers with liver cirrhosis, and the largest among the carriers with hepatocellular carcinoma, This iS because the number of caseS with low e-antigen units and of cases with co-existence of e-antigen and e-antibody increaSes in this order.
    4) The number of positive cases as determined by ID corresponded to that as determincd by RIA, except one case. However, 42% of cases considered as positive for eantigen by RIA and 51% of cases positive for e-antibody were judged as negative by ID. This suggested that RIA is more sensitive than ID.
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  • Clinical study of HBe-antigen, -antibody system
    Michio KATO, Takumasa OKUYAMA, Manabu MASUZAWA, Shuji HAZAMA, Shigeo M ...
    1980 Volume 21 Issue 10 Pages 1310-1315
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1310
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    1) In the patients with HBs-antigen positive liver disease (82 cases) and asymptomatic carriers (16 cases), 24 cases (24.5%) were HBe-antigen positive, 65 cases (66.3%) were HBeantibody positive and nine cases (9.2%) were both negative.
    2) In the subjects over forty years old (male 34 cases, female 16 cases), only two females (12.5%) were HBe-antigen positive, although nine cases (26.5%) in male.
    3) In the cases with more than 29 of HBs-antigen titer (r-PHA), significantly more subjects were HBe-antigen positive than in those with less than 28.
    4) HBe-antigen, -antibody seroconversion were found in 10 of 35 cases of chronic liver disease by serial follow up study. In six cases of them, seroconversion was detected over twice.
    5) These seroconversion cases had following tendency. When HBs-antigen titer reached its peak, HBs-antigen appeared. After almost one month the serum transaminase level reached its peak. With the descend of the HBs-antigen titer and the transaminase level, the next seroconversion from HBe-antigen to HBe-antibody occured. The same pattern of HBe-antigen, -antibody seroconversion was recurred serially in same cases.
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  • Relationship between lymphocyte suppressor activity on immunoglobulin production and presence of autoantibodies in patients with chronic liver disease
    Katsuaki SUGIURA, Yasushi HASUMURA, Jugoro TAKEUCHI, Junichi YATA
    1980 Volume 21 Issue 10 Pages 1316-1321
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1316
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    We Studied the relationship between concanavalin A-induced suppressor activity (Con ASA) of peripheral lymphocytes and presence of autoantibodies in patients with chronic liver disease.
    Among 21 patients studied, 5 had liver cirrhosis, 13 had chronic active hepatitis, and 3 had chronic inactive hepatitis. Eleven healty adults were used as controls. Peripheral blood lymphocytes prepared from the patients and from the controls were precultured with Con A for 2 days. Con A-activated lymphocytes were added to the fresh peripheral blood lymphocytes obtained from a normal donor and cultured for 7 days with Pokeweed mitogen. ConASA was calculated by counting the number of immnoglobulin-forming cells from these cultures.
    ConASA among normal controls were 100.9±15.0% (Mean±2SD). According to.this ConASA, patients with chronic liver discase were divided into three subgroups; normalactivity, low-activity, and high-activity group. Autoantibodie (i.e., anti-DNA antibody, antinuclear factor, rheumatoid factor, anti-smooth muscle antibody, and anti-mitochondrial antibody) were detected more frequently in sera from patients with low or high-activity than those with normal-activity. These results suggest that abnormalities of suppreSsor function are frequently observed in the patients with chronic liver disease and they are related to the appearance of autoantibodies.
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  • Mechanism for development of systemic endotoxemia in patients with cirrhosis and portal endotoxemia
    Kunihiko KOHNO, Naondo SUZUKI, Kunihiko OHMSHI, Hirotaka MUSHA, Kazuro ...
    1980 Volume 21 Issue 10 Pages 1322-1329
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1322
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    Twenty cirrhotic patients were studied fbr endotoxin both in splanchnic blood (portal vein, superior mesenteric vein, splenic vein) and peripheral vein blood.
    Endotoxin was positive in portal vein blood in 75% of the patients, in superior mesenteric vein blood in 85%, in splenic vein in 50% and in peripheral vein blood in 40%.
    In 8 of the 20 patients, endotoxin was positive in both splanchnic vein blood and peripheral vein blood (A group), and in 9, endotoxin was positive in splanchnic vein blood, but negative in peripheral vein blood (B group).
    ICG plasma retention rate as an index of hepatic dysfimction was significantly higher in group A (57±11%) than in group B (30±17%), the difference being significant (p<0.05).
    Intra hepatic shunt index was also higher in group A than in group B. However, there was no significant difference in the total- and extrahepatic shunt indices between the two groups.
    These findings suggested that reduced functional hepatic mass due to the intrahepatic shunting may play an important role in the development of systemic endotoxemia in patients with cirrhosis.
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  • Nutritional and pathophysiological studies on liver cirrhosis (Part one)
    Ingested amounts of various nutrients, especially amino acids, in the diet specially for cirrhotic patients and their nutritional assessments and pathophysiologies of the liver
    Misako OKITA, Akiharu WATANABE, Hideo NAGASHIMA
    1980 Volume 21 Issue 10 Pages 1330-1339
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1330
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    Actually ingested amounts of various dietary nutrients, especially amino acids, in 10 hospitalized and well compensated cirrhotic patients and their nutritional assessments and pathophysiologies of the liver were studied fbr investigating the possible dietetic therapy of cirrhotics. The liver diet specially for cirrhotics was fbund to be rich in protein but remarkably low in non-N energy/g N as compared to the ordinary diet for hospitalized patients without digestive diseases as control. Ingested amounts of energy and vegetable fat were significantly low in cirrhotic patients and molar ratios of branched chain amino acids (BCAA) to aromatic amino acids (AAA) showed lower values than those in the ordinary diet. Daily intakes of energy and protein in cirrhotic patients were correlated well with their nitrogen balances. BCAA/AAA in proteins, which cirrhotic patiehts take orally, showed the closest correlation to the severity of the disease as indicated by abnormal serum total protein and albumin concentrations and KICG values.BCAA and phenylalanine amounts and BCAA/(phenylalanine+tyrosine)in the ingested diet in cirrhotics also reflect those in the blood serum, respectively. Remarkably lower ratios of BCAA/ (phenylalanine+tyrosine)in serum were found in those patients, and significant correlation between this ratio and KICG values observed. The above results suggest that prescription of proteins containing high BCAA/AAA to cirrhotic patients may normalize abnormal serum BCAA/AAA and probably improve the impaired functions of the liver.
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  • Evaluation of computed tomography in the diagnosis of liver discases
    Katsumoto KATO, Tetsuo TAKAYAMA, Naoyuki KATADA, Daisaku NISHIMURA, To ...
    1980 Volume 21 Issue 10 Pages 1340-1351
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1340
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    In order to evaluate computed tomography (CT) in the diagnosis of liver disease, 90 cases of diffuse parenchymal diseases and 37 cases of mass lesions were examined with GE 8800 CT scanner. Abnormal CT findings in liver cirrhosis were characterized by splenomegaly, uneven liver margin and asites. Atrophy of right lobe and enlargement of left lobe could not be easily recognized on CT scan, compared with nuclear imaging. CT values of the liver were decreased and the ratios of CT values of the liver to those of the spleen were less than 0.9 in all cases with fatty liver. Jaundice in acute viral hepatitis can be easily differentiated from obstructive jaundice on CT scan because of observing no dilatation of intrahepatic bile duct. CT was superior in detecting space-occupying lesions to nuclear imaging and was more specific in that it was able to differentiate cystic from solid lesions. However, it was almost impossible to make a histological diagnosis of solid lesions even on CT scan.
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  • A case of fulminant hepatitis cured with the glucagon-insulin treatment
    Yukio HOSOKAWA, Mitsuo SUGA, Yoko YOKOYAMA, Hiroshi KINOSHITA, Tetsuro ...
    1980 Volume 21 Issue 10 Pages 1352-1357
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1352
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    A 17-year-old male patient with fulminant hepatitis who could be cured with the glucagon-insulin (G-I) treatment is reported. The patient was severely jaundiced (total scrum bilirubin 12.6mg/dl), his serum alanine aminotransferase 828 units, blood ammonia 101μg/dl and both HBs antigen and HBs antibody were simultaneously detected with the radioimmunoassay. Intensive parenteral administrations of prednisolone and l-DOPA, combined with rectal applications of lactulose, proved ineffective in preventing from his Grade III (Adams and Foley) hepatic encephalopathy with asterexis. The G-I treatment was therefore started, which resulted in awakening from coma with a decrease of blood ammonia. During the clinical course, serum AFP level was as high as 782ng/ml, and the liver biopsy specimen obtained after recovery showed cirrhosis of the liver. The mechanism by which the G-I treatment favours the fulminant hepatic disorder was discussed.
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  • Cytoplasmic blood plasma inclusions of hepatocytes associated with idiopathic portal hypertension-report of a case
    Kenji ABE, Tadao OKANO, Toshio SHIKATA, Yoji WAKU, Shoichi SHIONOME, Y ...
    1980 Volume 21 Issue 10 Pages 1358-1365
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1358
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    A case with cytoplasmic inclusions composed of blood plasma elements was reported. The patient was 9-year-old boy, who showed splenomegaly and esophageal varices with portal hypertension.
    Light microscopic examination of the biopsied liver revealed cytoplasmic eosinophilic inclusions in marked swelling hepatocytes. The inclusions were numerously scattered throughout liver lobules, mostly round in shape, variable in size, measuring 3μ to 20μ in diameter, and positive for diastase rcsistent PAS.
    It revcaled that most of the inclusions were positive for such blood plasma components as albumin, IgG, IgM, IgA, transferrin and fibrinogen by immunoperoxidase method. These inclusions are usually observed in the state of increase of intrasinusoidal blood pressure. Sclerosis of central vein and perisinusoidal fibrosis also noted. Thus, it was postulated that portal hypertension of this case was caused mainly by increase of intrasinusoidal blood pressure.
    To our knowledge, the present case represents the first report in which the exact nature of cytoplasmic blood plasma inclusions in human hepatocytes has been demonstrated by the technique of the immunoperoxidase method.
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  • An autopsy case of absence of the intrahepatic bile ducts
    Pathological study and comparison with the liver of congenital biliary atresia
    Hideo TERAO, Hideyo ITAKURA, Ichiro SEKINE, Jyoshu MATSUDA, Yoshiro TS ...
    1980 Volume 21 Issue 10 Pages 1366-1378
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1366
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    A post-mortem case of "Absence of the Intrahepatic Bile ducts" of a female infant of 1.5 years of age was presented.
    The Jaundice was noticed on the third day of life and persisted until death. "Congenital Extrahepatic Biliary Atresia" Was diagnosed clinically.
    Wedge biopsy specimen of the liver at 2.5 months of age revealed the presence of interlobular bile ducts in half of 14 portal triads examined and destructive or degenerative changes in different degrees of the bile ducts (i.e disarrangement of the epithelium and nuclei, degeneration, scarring and disappearing of the bile ducts). Liver cells showed ballooning degeneration with occasional giant cell transformation. Neither fibrosis or proliferation of bile ducts was observed.
    At autopsy, the extrahepatic biliary system was patent though hypoplastic. Intrahepatic bile ducts was completely absent; any trace of the bile duct could not be seen in all liver sections examined. Hepatic fibrosis and cholestasis were slight to moderate in centrilobular areas. However, there was no evidence of periportal fibrosis or proliferation of bile buct.
    These findings are apparently different from liver change of "Congenital Extrahepatic Biliary Atresia", and compatible with those of "Alagine Syndrome" and/or "Paucity of the Intrahepatic Bile Ducts".
    Intrauterine infection was suggested as its etiology.
    This report seems to be the first autopsy report of the disease in Japan.
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  • A case immunologically disturbed by thorotrast-induced hepatoma
    Shigeo BOKU, Michio KATO, Takumasa OKUYAMA, Manabu MASUZAWA, Shigeo MA ...
    1980 Volume 21 Issue 10 Pages 1379-1384
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1379
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    A case, 57 years old man, with thorotrast-induced hepatocellular carcinoma was reported. Thorium deposits in the reticuloendothelial tissues have the hazardous radiation effects on those tissues fbrever.
    In this case, the mean thorium amount in the spleen was 28.4% and the mean activity ratio of Ac-228 to Th-232 was 0.948. These values were much different from thoge reported by Kato in 1968.
    The ratio of T-cell to T.B. population decreased and PPD and PHA intradermal skin test were negative. Plasma cells in bone marrow increased and IgG was 4g/dl. In these immulogical studies it was supposed that T-cell function was impaired and B-cell function, especially IgG fraction was stimulated.
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  • A case of hepatocellular carcinoma with the skull metastases
    Daizo KAN, Mami KAN, Kinya MURATA, Toshio MISHIMA, Hiromi ONISHI, Taka ...
    1980 Volume 21 Issue 10 Pages 1385-1389
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1385
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    43-year-old man of hepatocellular carcinoma with bone metastases, especially to the skull was reported. There was no previous history of liver disease. He complained of severe headache, lumbago, back pain, leg pain, and right hypochondralgia. Liver function tests showed slight increase of alkaline phosphatase, γ-GTP, GOT and GPT and prominent decrease of cholinesterase. In serological tests, HBs antigen and HBs antibody were negative. The levels of alphafetoprotein and CEA were within normal. In radiology, many osteolytic areas were demonstrated. The present study indicates the multiplicity of syndrom in patient with hepatocellular carcinoma.
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  • [title in Japanese]
    [in Japanese]
    1980 Volume 21 Issue 10 Pages 1390
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1390
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese]
    1980 Volume 21 Issue 10 Pages 1391
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1391
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese]
    1980 Volume 21 Issue 10 Pages 1392
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1392
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1393
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1393
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1394
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1394
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1395
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1395
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1396
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1396
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1397
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1397
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1398
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1398
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese]
    1980 Volume 21 Issue 10 Pages 1399
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1399
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1400
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1400
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1401
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1401
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1402
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1402
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1403
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1403
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese]
    1980 Volume 21 Issue 10 Pages 1404
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1404
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1405
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1405
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1406
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1406
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese]
    1980 Volume 21 Issue 10 Pages 1407
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1407
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1408
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1408
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1980 Volume 21 Issue 10 Pages 1409
    Published: October 25, 1980
    Released on J-STAGE: May 26, 2009
    DOIhttps://doi.org/10.2957/kanzo.21.1409
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  • [title in Japanese]
    1980 Volume 21 Issue 10 Pages 1410-1429
    Published: October 25, 1980
    Released on J-STAGE: January 19, 2010
    DOIhttps://doi.org/10.2957/kanzo.21.1410
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