Kanzo
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Volume 28, Issue 6
Displaying 1-24 of 24 articles from this issue
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  • 2'-5'oligoadenylate synthetase activity in serum and PBMC of chronic hepatitis B on interferon treatment
    Michiko SHINDO, Tadao OKUNO, Masayuki MATSUMOTO, Makoto TAKEDA, Yoshim ...
    1987 Volume 28 Issue 6 Pages 675-680
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.675
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    Ten HBsAg- and HBeAg-positive patients with chronic active hepatitis, all of whom were treated with interferon (IFN) were studied for serial measurement of 2'-5'oligoadenylate synthetase (2-5AS) activity in serum and peripheral blood menonuclear cells (PBMC). Ten healthy adults and 10 HBsAgand HBeAg-positive chronic hepatitis patients who hadn't received antiviral treatment, were also studied. IFN was given daily for 4 weeks in 6 and intermittently in 4 (1 week administration followed by 1 week pause with 4 cycles). 2-5AS activity elevated rapidly on IFN administrations and fell on its discontinuance. 2-5AS activity had an inverse correlation with DNA-P, which we called a mirror image phenomenon. These suggested 2-5AS activity in serum and PBMC would be a reliable marker of antiviral action of IFN. 2-5AS in serum had a good correlation with that in PBMC (r=0.875). Therefore, we may conclude that 2-5AS in serum would be substitutive for that in PBMC and a reliable marker of antiviral action of IFN on IFN treatment.
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  • A study of superoxide dismutase activity in the liver tissue and the serum of the patients with viral hepatitis
    Hitoshi TOGASHI, Haruhide SHINZAWA, Hiroto WAKABAYASHI, Nobuo YAMADA, ...
    1987 Volume 28 Issue 6 Pages 681-686
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.681
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    Superoxide dismutase activities (SOD) in the liver tissue and the serum of patients with viral hepatitis were measured. SOD were assayed according to Oyanagui's method.
    Lipid peroxides (LPO) were also assayed according to Ohkawa's method. The liver total SOD, Cu, Zn-SOD and Mn-SOD activities in patients with acute hepatitis were significantly higher than those in normal controls. The liver SOD in chronic liver diseases were lower than those in normal controls. As to chronic liver diseases, SOD activities especially cytosolic Cu, Zn-SOD decreased according to the progress of the diseases. Between SOD activities and LPO in the liver tissue, there were positive correlations in acute, chronic hepatitis and liver cirrhosis. Particularly, a significantly positive good correlation between them was noted in chronic active hepatitis. These findings suggest that SOD were induced remarkably by oxygen radicals in acute hepatitis, but in chronic liver diseases the extent of SOD induction by oxygen radicals were declined in accordance with the progress of tissue injury. On the other hand, level of the serum SOD activities increased in liver diseases. This might be due to the release of SOD into the blood from the continuous damaged liver cells by oxygen radicals.
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  • Protective effect of dibutyryl adenosine 3', 5'-monophosphate on chronic liver damage induced by carbon tetrachloride in the rat
    Kohdoh ISHII, Shigenori NII, Hitomi KARUBE, Yoshikuni FUJITA, Hisao SH ...
    1987 Volume 28 Issue 6 Pages 687-691
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.687
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    An effect of dibutyryl adenosine 3'-5'-monophosphate (DBcAMP) on chronic liver injury induced by carbon tetrachloride (CCl4) was investigated. Fifty male Wistar rats were divided into the following groups: (1) controls (n=20), treated with CCl4 only; (2) rats treated with DBcAMP simultaneously with CCl4 (n=30). CCl4 (0.2ml/100g bw) was injected subcutaneously twice weekly, and DBcAMP (1.0 mg/100g bw)was administered intraperitoneally twice daily and six days weekly. All animals receiving CCl4 alone showed parenchymal cell damage and hepatic fibrosis within a short period of time, which led to liver cirrhosis and died of hepatic failure within 15 weeks of treatment. Microangiograms represented conspicuous rarefaction of the intrahepatic vascular beds and disordered vascular contour, Animals treated with DBcAMP, on the other hand, showed neither rarefaction of the vascular beds nor atrophy of the liver and all animals survived even consecutive CCl4 exposure for 17 weeks. It is concluded that in protecting microcirculatory system of the liver, DBc-AMP provides potent preventive effect on the chronicity of liver damage induced by CCl4.
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  • Experimental studies of blood-brain barrier permeability, transport of amino acids across blood-brain barrier and brain edema in acute liver failure
    Michio KOBAYASHI
    1987 Volume 28 Issue 6 Pages 692-702
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.692
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    Time course of amino acids of plasma and cerebrospinal fluid (CSF), the permeability of bloodbrain barrier (BBB) and brain edema were studied using animal model such as galactosamine (GS) induced rat liver failure, and the effects of predonisolone (PS) were evaluated. After GS administration, the concentration of amino acids gradulally elevated in both plasma and CSF, but the degree was marked in plasma. The permeability of the BBB, which was measured using radioactive L-Glucose, increased following GS injection, and reached to 250 percent of control at 48 hours. Intake of amino acids into brain slightly increased, which was significantly higher in branched chain amino acids than in aromatic amino acids. In PS treated rats before GS, the increase of amino acids in plasma and CSF was lower than that without PS. Same result was obtained about the permeability of the BBB. When PS was given after GS, no remarkable change was observed. However, PS after GS also significantly prevented the brain edema. These results lead to conclude that assessment of early indication of PS in acute liver failure needs further investigation.
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  • Ultrastructural changes of intermediate filaments of hepatocyte in alcoholic liver disease
    Hisataka OGASAWARA, Takashi TSUCHIYA, Hiroyo OHISHI, Fumi MIZOROGI, Ts ...
    1987 Volume 28 Issue 6 Pages 703-711
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.703
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    This study was aimed to clarify the ultrastructural changes of intermediate filaments (IFs) of hepatocyte in alcoholic liver disease. Ten patients with alcoholic liver disease, five patients with normal liver and five patients with viral liver diseases were subjected to this study. Liver biopsies were undergone under laparoscopy or at laparotomy. Small pieces of the liver tissue about 1cmm in size were soaked in stirring buffer solution containing 0.5% Triton X-100 for 90min at 37°C. The tissues were fixed with 2% OsO4 for 3 hours at room temperature and then were dehydrated in ethanols and embedded in epoxy resin. Ultrathin sections were observed with transmission electron microscope. IFs 6-11nm in diameter were distributed evenly in normal liver. On the other hand IFs were distributed unevenly and some of them were thickened up to 11-20nm in diameter in alcoholic liver disease. The continuity of IFs to some component filaments of Mallory body was observed. No apparent changes of IFs were seen in viral liver diseases. It was considered that these findings of IFs were useful for clarifying pathogenesis and diagnosis of alcoholic liver disease.
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  • Effects of a calcium channel blocking agent, diltiazem, and calciumionophore (A23187) on hepatic cell injury by CCl4 in rat cultured hepatocytes
    Shohichi HASEGAWA, Shigeo TSUYAMA, Bungo NISHII, Keizoh KAWANO, Seiji ...
    1987 Volume 28 Issue 6 Pages 712-718
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.712
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    The effects of a calcium channel blocking agent, diltiazem, and calciumionophore (A23187) on hepatic injury and 45Ca uptake to hepatocytes induced by CCl4 in cultured rat hepatocytes were studied. Consequently, the following results were observed.
    Addition of CCl4 to cultured rat hepatocytes increased enzyme release, such as GOT and LDH, into the medium and 45Ca uptake to hepatocytes. Treatment with diltiazem inhibited these enzyme release and 45Ca uptake, while treatment with A23187 reversed the changes.
    These findings suggest that diltiazem may protect against liver cell injury induced by CCl4 accompanied with the inhibition of Ca uptake to hepatocytes.
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  • The study on the chronicity of experimental autoimmune hepatitis in neonatally thymectomized mice
    Yasuyuki WATANASE, Kazunari MASUDA, Yoshihiro IKEMOTO, Hiroiku KAWAKAM ...
    1987 Volume 28 Issue 6 Pages 719-726
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.719
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    Using an autoimmune hepatitis model of A/J mice which was prepared with immunization of syngeneic liver proteins, we observed influence of neonatal thymectomy on the chronicity of hepatitis. Hepatitis was severer in neonatally thymectomized mice (group B) than in non-thymectomized controls (group A). 3 months after the final immunization, piecemeal necrosis was recognized with high frequency in group B but it was not recognized in group A. The level of autoantibody to liverspecific lipoprotein (LSP) was higher in group B than group A, and high level of that in group B continued unchangeably until 3 months after the final immunization. Production of the autoantibody to LSP was reduced by adoptive transfer of normal mouse spleen cells, but the spleen cells of neonatally thymectomized mice showed no such effects. This experiment suggests that neonatal thymectomy is apt to abolish tolerance to LSP on account of depressed suppressor activity to autoantigen, and accordingly hepatitis is increased and continues.
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  • Lymphokine-activated killer activity and natural killer activity in patients with hepatocellular carcinoma
    Hiroshi SAKAEDA, Toshiji SAIBARA, Saburo ONISHI
    1987 Volume 28 Issue 6 Pages 727-734
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.727
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    Lymphokine-activated killer (LAK) activity and natural killer (NK) activity in patients with hepatocellular carcinoma (HCC) were assessed and proven to be severely impaired in comparison with those in healthy volunteers and liver cirrhosis (LC). NK activity, or LAK activity in individual HCC patients could not be correlated with age, size of the tumor mass, and liver function tests. The sera of patients with HCC showed inhibitory effect on NK activity and on the induction of LAK activity from normal peripheral mononuclear cells, but this serum inhivitory activity on LAK induction was abrogated by 56°C, 30 minutes heat-inactivation or by adding an increased amount of rIL-2 in the culture medium.
    So immune surveillance on oncogenesis is shown to be impaired, and suppressed by heat-labile serum inhibitor in HCC patients.
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  • Therapy of hepatocellular carcinoma complicated with intravascular tumor emboli
    Effectiveness of radiotherapy for tumor emboli
    Tohru NAGASMHIMA, Munemasa RYU, Minoru MUKAI, Takamitsu ARIGA, Zaikan ...
    1987 Volume 28 Issue 6 Pages 735-744
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.735
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    Radiotherapy was performed for 12 cases of hepatocellular carcinoma complicated with tumor emboli, and the results were evaluated. Irradiated dose was, in principle, Time-Dose-Fractionation factor (TDF) 80 and radiation was performed for tumor emboli only, TAE was performed for main tumors simultanously. After radiation, all of the cases showed a reduction of tumor embolus on medical imagings. No cases showed that the tumor embolus had expanded. Histologically, 80% of the cases revealed an effectiveness of greater than grade IIA under Ohboshi's and Shimosato's classification. By CT or liver scintigraphy, atrophy of the irradiated field and compensatory hypertrophy of the non-irradiated field were observed.
    In the unresected cases of hepatocellular carcinoma complicated with tumor emboli, irradiated group was obtained better prognosis than unirradiated another therapy group.
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  • Comparative changes of alterial blood keton body ratio and hepatic function induced by transcatheter arterial embolization and intra-arterial injection of lipiodol-adriamycin suspension in patient with liver cancer
    Munemasa RYU, Hiroshi YAMAMOTO, Hiroyuki HIRASAWA, Minoru MUKAI, Takam ...
    1987 Volume 28 Issue 6 Pages 745-754
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.745
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    Arterial blood keton body ratio (AKBR) reflects an energy charge in the cells of the liver and is a sensitive parameter to learn functional profile of the liver. Therefore, we measured the value to estimate changing aspects of the liver function after transcatheter arterial embolization (TAE) and intra-arterial injection of lipiodol-adriamycin suspension (LA) in patients with liver cancer.
    Seventy-one patients were treated with the following four group modalities. 1) C-TAE group (15 patients); TAE was carried out using sliced gelfoam pieces after intra-arterial administration of mitomycin-c (Conventional TAE), 2) L-TAE group (35 patients); TAE was completed using sliced gelfoam pieces after intra-arterial injection of LA, 3) LA group (17 patients); LA was achieved via hepatic artery, and 4) control group (4 patients); Only hepatic arteriography was performed.
    In patients of both C-TAE and L-TAE groups AKBR decreased immediately after treatment; in a half of them the value decreased continuously for two hours and increased 24 hours later. Changes of the liver function parameter were temporary in the majority of patients treated with these modalities. However, AKBR in some patients associated with severe liver cirrhosis, jaundice or markedly extended liver carcinoma decreased below 0.3 after TAE and these patients died of hepatic insufficiency. On the other hand, AKBR did not decrease in LA group and showed an increasing tendency two hours later. Moreover, no remarkable changes in liver function was induced on LA group and LA was also safe in patients with severe liver cirrhosis, jaundice or liver carcinoma mass in a large size.
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  • Clinical and pathological studies on hepatic fungus infections
    Shinji YAMADA
    1987 Volume 28 Issue 6 Pages 755-765
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.755
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    A statistical survey of hepatic fungus infections in Japanese pathological autopsies (1972-1981) was carried out. The incidence was 0.19% (521 cases) in the total 280, 532 autopsies, and 7.4% in overall fungus infections. Most of them were assumed to be partial phenomena of systemic fungus infections. The predominant type of infection was candidiasis.
    Thiry-five autopsy cases with hepatic fungus infections in our department were submitted for clinical and pathological investigations. Overt jaundice, increased S-GOT and S-GPT values were common clinical findings of cases.
    Macroscopically, capsulitis and multiple nodular lesions were the frequent evidence, and the latter was revealed in about half of the cases (51.4%) by surface inspection of the liver. By tissue reaction, the fungal lesions were classified into coagulation necrosis type (77.1%) and abscess-forming type (11.4%). It was concluded that coagulation necrosis type was the common lesion among the patients with severe immuno-deficiency, and abscess-forming type was frequent in immuno-resistant hosts, the infection route, and morphogenesis of the lesions were studied and the results were discribed.
    It was also pointed out that rare incidence of hepatic fungal lesions in liver cirrhosis, and the reason was discussed.
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  • A male case of primary biliary cirrhosis (PBC) complicated with lupoid hepatitis
    Sekio MOTEKI, Koichi SAITO, Nobuo SAITO, Yukio SATO, Kazuei ORIKASA, K ...
    1987 Volume 28 Issue 6 Pages 766-771
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.766
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    A male case of primary biliary cirrhosis (PBC) with lupoid hepatitis is reported. A 53-year-old man was admitted to our hospital because of liver dysfunction. Laboratory data showed high levels of serum Al-p (22 KAU), LAP and y-GTP (387mU/ml), and amount of serum IgG elevated extremely up to 3566mg/dl. Anti mitochondrial antibody (AMA), and smooth muscle antibody (ASMA), anti nuclear antibody (ANA), and DNA and anti SS-B were positive. Using extracts of bovine heart mitochondria as a source of antigen, patients serum such as other PBC sera showed two precipitating lines by double immunodiffusion test. However, the subtype of AMA was anti-M2, not anti-M4.
    Histological examination of surgical biopsy showed typical chronic non-suppurative destructive cholangitis (CNSDC), enlarged portal tracts with heavy imflammatory cells mainly composed of lymphocytes and plasma cells, and piecemeal necrosis characterized by chronic active hepatits (CAH). The levels of ANA, anti-DNA and transaminase were well responded to steroid therapy.
    Therefore, this case was diagnosed as overlapping case of asymptomatic PBC and lupoid hepatitis although anti-M4 was not detected. It would be difficult to diagnosis CAH-PBC mixed form by reason of positive or negative anti-M4.
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  • A case of lupoid hepatitis associated with hepatocellular carcinoma
    Seiya ARIMA, Ken YOSHIDA, Osamu HASHIGUCHI, Shunichi KIMURA, Seiichi O ...
    1987 Volume 28 Issue 6 Pages 772-777
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.772
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    A 60-year-old woman with hepatocellular carcinoma about 7 years after the diagnosis of lupoid hepatitis, was reported.
    Her liver dysfunction was pointed out in 1976, and icterus and ascites developed in 1978. Laboratory examinations showed hypergammaglobulinemia (5.0g/dl), 6350mg/dl of IgG, and positive LE cell, antinuclear antibody and anti-DNA antibody. Diagnosis of lupoid hepatitis was made. Symptomatic relief was obtained after about two years of steroid therapy. Laparoscopy in October, 1979 revealed nodular liver and chronic active hepatitis was confirmed histologically.
    In October, 1985, high AFP and various image diagnosis confirmed the complication with hepatocellular carcinoma, which led to her death due to intraperitoneal massive hemorrhage in March, 1986. Needle necropsy revealed findings of liver cirrhosis (Nagayo's type A) and trabecular-type hepatocellular carcinoma of Edmondson Type II. So far as we know, this case was the 7th one in the world.
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  • A case of hepatocellular carcinoma accompanied by adenomatous hyperplasia and borderline lesion of hepatoma pathologically
    One of the tumors was deposited with lipiodol and the other was not with lipiodol simultaneously
    Hitoshi TAKAGI, Shoji YAMADA, Yoshihiro OHNO, Takehiko ABE, Takashi TA ...
    1987 Volume 28 Issue 6 Pages 778-784
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.778
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    55 year old male admitted to our hospital because of the elevation of serum a-fetoprotein and hypoechoic mass on ultrasonography. He had been followed up as liver cirrhosis with positive HBsAg. Two hypervascular tumors with diameter of 2cm×3cm were found in right lobe on angiography, transcatheter arterial embolization (TAE) with Adriamycin + Lipiodol was done twice. Blood transfusion was needed for bleeding from duodenal ulcer after TAE and transcatheter splenic arterial embolization (TSAE) was done as preoperative treatment for the improvement of hypersplenism. One month after TSAE, hepatic failure became worsen and died three month after admission. Lipiodol was deposited to one tumor and not to the other one on computed tomography. Microscopically, the former was completely necrotic, and the other was viable, containing hepatocellular carcinoma (Edmondson II). Adenomatous hyperplasia containing the borderline lesion of hepatocellular carcinoma were found in the other part of the liver. These findings seems to support the multicentric origin theory of hepatocellular carcinoma.
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  • A case of multiple liver cell adenoma
    Nobuaki HIRATA, Masao KIM, Makoto NISHIYAMA, Hiroki AKAMATSU, Akira KI ...
    1987 Volume 28 Issue 6 Pages 785-793
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.785
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    Liver cell adenoma is very rare tumor. We experienced a case of multiple adenomas of the liver. A 14-year-old girl was admitted for the purpose of the examination of anemia. Extensive clinical investigation such as ultrasonography, CT scan, angiography, and scintigraphy, revealed multiple tumors in the liver. The needle biopsy was failed, and open biopsy was performed. A tumor in the left lobe was extirpated, and the diagnosis of liver cell adenoma was taken. 15 cases of multiple adenomas of the liver were reported in the world, and 4 cases in Japan. Malignant transformation of liver cell adenoma was reported in a few literatures. In this case, the adenomas were gradually enlarged, so we must follow up this case strictly.
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  • A case of primary hepatic lymphoma with cirrhosis of the liver
    Junich INOUE, Masahiro NOMURA, Shoso NAKAGAWA
    1987 Volume 28 Issue 6 Pages 794-800
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.794
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    A 55-year-old woman was pointed out abnormal liver function tests in July 1979 and a diagnosis of chronic hepatitis (non B) was established. 99mTc-Scintigram showed a space occupying lesion in the right lobe of the liver in August 1982. Physical examination revealed hepatomegaly. Liver function tests showed high values of a lactic dehydrogenase (1088mIu/ml) and a ferritin (300ng/ml). The imaging diagnoses demonstrated hepatocellular carcinoma with cirrhosis of the liver. Although she was treated with the immunochemotherapy, she died of hepatic failure in March 1983. The autopsy disclosed cirrhosis of the liver (Z type) and a large yellowish gray tumor (14×10×8cm). Microscopic findings of the tumor showed tumor cells were separated to each other. The electron microscopy and the peroxidase antiperoxidase method diagnosed the case as primary hepatic lymphoma, which was diffuse lymphoma of the large cell type (non-cleaved, B cell) in the Lymphoma Study Group.
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  • Serum procollagen III peptide (Fab) in various liver diseases
    Kenji KOJO, Shinichiro YAMAMOTO, Ituro SAITO, Ryosuke YAMAMOTO, Seiji ...
    1987 Volume 28 Issue 6 Pages 801-802
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.801
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  • Increase of type I procollagen m-RNA of human fibroblast induced by mononuclear cells from the patient with chronic active hepatitis
    Koetsu MORITA, Yoshiro NIITSU, Nobuyuki ITO, Minoru OWADA, Kunihiko MA ...
    1987 Volume 28 Issue 6 Pages 803-804
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.803
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese]
    1987 Volume 28 Issue 6 Pages 805
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.805
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1987 Volume 28 Issue 6 Pages 806
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.806
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    1987 Volume 28 Issue 6 Pages 807
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.807
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese]
    1987 Volume 28 Issue 6 Pages 808
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.808
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  • [title in Japanese]
    [in Japanese], [in Japanese], [in Japanese], [in Japanese]
    1987 Volume 28 Issue 6 Pages 809
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.809
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  • [title in Japanese]
    1987 Volume 28 Issue 6 Pages 810-851
    Published: June 25, 1987
    Released on J-STAGE: July 09, 2009
    DOIhttps://doi.org/10.2957/kanzo.28.810
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