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Volume 44, Issue 3
Displaying 1-5 of 5 articles from this issue
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Now and FutureHiroshi Okada2002 Volume 44 Issue 3 Pages 241-253
Published: September 28, 2002
Released on J-STAGE: August 25, 2010
JOURNAL FREE ACCESSDownload PDF (4905K) -
Shogo Takashiba2002 Volume 44 Issue 3 Pages 254-260
Published: September 28, 2002
Released on J-STAGE: August 25, 2010
JOURNAL FREE ACCESSDownload PDF (1072K) -
Akiko Hisano, Hiroshi Nakaya, Tsutomu Sato, Kyuichi Kamoi2002 Volume 44 Issue 3 Pages 261-272
Published: September 28, 2002
Released on J-STAGE: August 25, 2010
JOURNAL FREE ACCESSWe studied risk indicators in saliva for periodontal disease in the elderly. Thirty-six subjects over 65 years old (15 men and 21 women) were divided into 4 groups based on clinical evaluation: 7 with healthy gingiva or gingivitis, 11 with moderate periodontitis, 9 with severe periodontitis, and 9 edentulous subjects. Whole saliva samples were analyzed for the presence of Actinobacillus actinomycetemcomitans, Porphyromonas gingivalis, Prevotella intermedia, Bacteroides forsythus, and Treponema denticola using polymerase chain reaction (PCR) method. We studied the relationship of periodontal parameters to the presence of these periodontopathic bacteria, lactate dehydrogenase (LDH), aspartate aminotransferase (AST), and interleukin-1 (IL-1) gene polymorphism. The prevalence of Bacteroides forsythus in subjects with severe periodontitis was significantly higher than in those with healthy gingiva or gingivitis (p<0.05). LDH activity increased proportionally with the advance of periodontitis (p<0.05). AST and IL-1 polymorphism did not correlate significantly with periodontal status. This suggests that the presence of Bacteroides forsythus and LDH activity in whole saliva are possible to be risk indicators for periodontal disease in the elderly. J Jpn Soc Periodontol 44 : 261-272, 2002.View full abstractDownload PDF (1643K) -
Hiroshi Nitta, Shigeru Oda, Daiji Furuse, Isao Ishikawa2002 Volume 44 Issue 3 Pages 273-280
Published: September 28, 2002
Released on J-STAGE: August 25, 2010
JOURNAL FREE ACCESSGracey curette blade adapts appropriately to the root surface and minimizes damage to hard and soft tissues during scaling and root planing. Sharpening should produce a sharp cutting edge without altering the original design.
We reevaluated curette blade design and propose a new sharpening technique based on this design. Five different Gracey curettes (# 1, # 5, # 7, # 11, and # 13) from 2 manufacturers (Hu-Friedy Co. Ltd. and Nordent Co. Ltd.) were evaluated. The terminal shank of the curette was held vertical to the floor. Images of blades from a point above the face were taken at×40 magnification by a digital stereomicrocope. Lateral blade images were also taken with the terminal shank of the curette held horizontal to the floor. Curvature of the cutting edge was determined from both images using computer software.
Results indicated that the cutting edge of all curettes was a straight line from the shank end to the toe in images from above the face. The lateral view showed that all blades had curved faces. Based on blade design, we proposed a new sharpening technique using 2-dimensional movements of both the instrument and sharpening stone. This technique is simpler than conventional sharpening in which only the sharpening stone is moved and complicated 3-dimensional sharpening strokes are required.
The proposed technique is simple, theoretically correct from a dimensional point of view, and applicable to any Gracey curette from different manufacturers. The technique diminishes the risk of distorting the original blade design. J Jpn Soc Periodontol, 44: 273-280, 2002.View full abstractDownload PDF (3331K) -
Takashi Azuma2002 Volume 44 Issue 3 Pages 281-291
Published: September 28, 2002
Released on J-STAGE: August 25, 2010
JOURNAL FREE ACCESSWe evaluated the role of interleukin-17 (IL-17) in the progression of periodontitis focusing on 1) the presence of mRNA expression of IL-17 and T cell cytokines in periodontal tissues, evaluated using RT-PCR and direct sequencing, IL-17 production in gingival crevicular fluid (GCF) by ELISA and cultured periodontitis tissues by Western blot analysis ; and 2) whether recombinant human IL-17 (rhIL-17) and culture supernatants of periodontitis tissues induced IL-6 production from cultured human gingival fibroblasts (HGF). We detected IL-17 mRNA expression in periodontitis tissues, detected by Western blotting, IL-17 in GCF samples was not detectable. T cells in periodontitis tissues simultaneously expressed mRNA encoding IL-17 and mRNAs of IFN-γ, IL-2, osteoclast differentiation factor (ODF) and osteoclastgenesis inhibitory factor (OCIF), which belong to Th 1 cytokines or bone metabolic molecules (Fisher's exact test), whereas the mRNA of IL-4, which is a Th 2 cytokine, was not expressed rhIL-17 induced IL-6 production by HGF in time- and dose-dependently. These findings indicate that activated T cells in periodontitis lesions produced IL-17, a Th 1 cytokine and bone metabolic molecule that may be responsible for enhancing inflammatory reactions and bone resorption via gingival fibroblast-derived mediators in periodontal disease. J Jpn Soc Periodontol 44: 281-291, 2002.View full abstractDownload PDF (2090K)
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