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全文: "流血"
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  • 塩澤 幸吉
    日本鍼灸治療学会総会論文集
    1954年 3 巻 1 号 15-16
    発行日: 1954/10/20
    公開日: 2011/05/30
    ジャーナル フリー
  • 梁瀬 恵子, 今村 貞夫
    日本皮膚科学会雑誌
    1981年 91 巻 5 号 549-
    発行日: 1981年
    公開日: 2014/08/21
    ジャーナル 認証あり
    皮膚科領域における自己免疫疾患およびその近縁疾患に対して血小板凝集法と Clq 結合試験による血中 Immune Complex (以下IC)の測定を試みた.膠原病の中では全身性エリテマトーデス,進行性全身性硬化症に高値の IC が認められ,とりわけ全身性エリテマトーデスでは血中 C3 値は低下がみられた.水疱性疾患のうちでは天疱瘡に高値の IC がみとめられた.紅斑および血管炎のうちでは皮膚血管炎,結節性紅斑で高値の IC を認めたが,多形溶出性紅斑・薬疹などにもしばしば IC が検出された.IC の多寡との間に一定した関係はみられないもののこれらの症例の3~6割に皮疹部に免疫グロブリンあるいは補体の沈着がみとめられた.血中 IC が高率に検出される SLE ,天疱瘡,結節性紅斑では臨床経過や他の検査データーとかなり相関して血中 IC も変動をみた.また皮膚筋炎・類天疱瘡・ DLE ・ジューリソグ疱疹状皮膚炎・ベーチェット病においては血小板凝集法では正常範囲内にとどまったが Clq 結合試験においては IC を検出し得た.検出し得る IC が方法によって異なる可能性があるが,いずれにしても血中 IC がいわゆる自己免疫疾患のみならず,その近縁疾患においても病因としてかなり重要な要素であることが想像された.
  • 岡村 潔
    日本藥物學雜誌
    1941年 33 巻 4 号 485-503
    発行日: 1941/12/20
    公開日: 2010/07/09
    ジャーナル フリー
  • 橋場 邦武
    日本循環器學誌
    1958年 21 巻 11 号 537-544
    発行日: 1958/02/20
    公開日: 2008/04/14
    ジャーナル フリー
    Nervous control of the coronary circulation has long been studied, but many problems remain to be further investigated. In general, the coronary blood flow increases by the sympathetic nerve stimulation, and decreases by the vagal nerve stimulation. But, the stimulation of the sympathetic or vagal nerve to the heart produces significant changes in hemodynamic and metabolic factors, such as blood pressure, heart rate, vigor of contraction, ratio of systole/diastole, cardiac output, extravascular compression, massging action of the heart, oxygen consumption and metabolites. Therefore, it can not be simply concluded that the sympathetic nerve causes active coronary vasodilatation, while the vagal nerve causes active coronary vasoconstriction.In the present paper, the effects of the stimulation of the stellate ganglia upon the coronary blood flow were reported (in the next paper, the stimulation of the vagal nerve is to be reported). Experimental methods were same as already described in Part I of this study.
  • 竹屋 範英
    日本薬理学雑誌
    1958年 54 巻 1 号 82-89
    発行日: 1958/01/20
    公開日: 2010/07/09
    ジャーナル フリー
  • 佐藤 邑太, 菊池 司
    画像電子学会研究会講演予稿
    2017年 16.04 巻 16-04-92
    発行日: 2017年
    公開日: 2020/07/01
    会議録・要旨集 認証あり
    近年、アニメ作品で 3DCG が様々な形で使用されている。しかし、そのまま作品に取り入れてしまう と違和感を与えてしまう。そこで、セルルックと呼ばれるセルアニメ風に表現する手法の研究が進められている。 そこで本研究は、多くのアニメ作品で目にすることのできる「過剰な流血シーン」に注目し、3DCG によるセルル ックな流血の生成手法を提案する。本研究では最初に血液の性質を調査し、それを基に FLIP 法で流体の動きを計 算して、リアルな流血のシミュレーションを行う。そして、生成されたシミュレーション結果にセルルックにする ための要素を加えて、セルルックな流血表現を実現させる。
  • 竹屋 範英
    日本薬理学雑誌
    1958年 54 巻 1 号 75-81
    発行日: 1958/01/20
    公開日: 2010/07/09
    ジャーナル フリー
  • 橋場 邦武
    日本循環器學誌
    1958年 21 巻 12 号 662-668
    発行日: 1958/03/20
    公開日: 2008/04/14
    ジャーナル フリー
    In the present patper, the effects of the stimulation of the peripheral end of the vagal nerve were reported, with consideration of the relationship between vasomotor and hemodynamic factors. Experimental methods were same as already described in the Part I of this study.Results were as follows : 1) Stimulations of the peripheral ends of the cut vagal nerves were performed, on the left side in 15 experiments on 11 dogs, on the right side in 4 experiments on 4 dogs. During the stimulation of the vagal nerve, the depression of the blood pressure and the bradycardia occurred in all experiments.2) Coronary blood flow decreased with the depression of the blood pressure, for example, as shown in Fig. 1. But, in about a half cases of all experiments, coronary blood flow increased initially for a short time, and then decreased with marked depression of the blood pressure, for example, as shown in Fig. 2 and 3. Experimental results of all cases in the initial increase and maximal decrease in coronary blood flow, were shown in Tab. I and II, respectively.3) Coronary vascular resistance decreased markedly during phases of the initial increase in coronary blood flow (white dots in Fig. 4). In addition, it decreased also during phases of the maximal decrease in coronary blood flow, in over a half cases of experiments (black dots in Fig. 4). Changes in coronary vascular resistance in the vagal nerve stimulation (Fig. 4) were quite different from that in the depressor response by the stimulation of the proximal end of the vagal nerve (Fig. 5) (already discussed in Part I of this study). It suggests coronary vasodilatory actions by the stimulation of the peripheral end of the vagal nerve.4) It seems likely that, the more decreases the heart rate, the more decreases the coronary vascular resistance, when the degree of the bradycardia is not over approximatery 40 %, but, in marked bradycardia, the coronary vascular resistance tended to increase, as shown in Fig. 6.The initial increase in coronary blood flow (white dots in Fig. 6), however, occurred with either very slight or marked bradycardia, in spite of depression of the blood pressure. It suggests the vagal active vasodilatation.5) From results reported in this paper, it may be concluded that ; the vagal nerve stimulation produces the coronary vosodilation and the initial increase in coronary blood flow ; but, the coronary blood flow decreases thereafter, or throughout during the stimulation from its beginning, because of the marked depression of the blood pressure ; the bradycardia itself may have some vasodilatatory effects on the coronary blood flow.
  • 鷲見 謙一, 相原 信幸
    日本内科学会雑誌
    1926年 14 巻 9 号 775-778
    発行日: 1926/12/10
    公開日: 2008/06/12
    ジャーナル フリー
  • 橋場 邦武
    日本循環器學誌
    1957年 21 巻 9 号 433-440
    発行日: 1957/12/20
    公開日: 2008/04/14
    ジャーナル フリー
    It has been widely known that the stimulation of the proximal end of the vagal nerve is followed by circulatory and respiratory reflexes. On the other hand, it has been suggested that reflexes to the coronary vessels from the visceral organs such as stomach, gallbladder and from the carotid sinus are carried through the cervical vagal nerve. In the persent paper, changes in coronary blood flow by the stimulation of the proximal end of the vagal nerve are reported.Coronary blood flow was measured by rotameter, in anesthetized (with sodium thiopental, 0.03-0.05g/kg intravenously) open-chest dogs weighing 10-15 kg. As shown in Fig. 1 (A), a speically designed cannula (Fig. 1 (B)) was inserted into the ostium of the left coronary artery, thus, a self-perfusing system to the left coronary artery from the right femoral artery was established. A typical calibration curve with rotameter was shown in Fig. 2. The calculated coronary vascular resistance was led from the relationship CVR=(BP)^^-/CBF, where CVR represents coronary vascular resistance, (BP)^^-, means blood pressure and CBF, coronary blood flow. The electrical stimulation of the nerve was carried out with spyky wave produced by thyratron. It was continued for 10-15 seconds with frequency of 15-30 cycles per second and intensity of 0.5-30 volts.
  • 中村 信雄
    日本傳染病學會雜誌
    1926年 1 巻 3 号 276-277
    発行日: 1926年
    公開日: 2011/11/25
    ジャーナル フリー
  • 相川 崇史, 谷本 潔昭, 堀内 淑彦
    アレルギー
    1980年 29 巻 7 号 446-
    発行日: 1980/07/30
    公開日: 2017/02/10
    ジャーナル フリー
  • 村田 和彦
    日本循環器學誌
    1959年 22 巻 11 号 814-821
    発行日: 1959/02/20
    公開日: 2008/04/14
    ジャーナル フリー
    The nervous control of the coronary circulation has been widely investigated, but many problems still remain unsolved. It is generally accepted that the coronary vessels are dilated by the sympathetic nerve and constricted by the vagal nerve. However, recent work of our clinic stands against these former concept. In this paper, for further study of the nervous control of the coronary vessels from pharmacological standpoint, the effects of some autonomic drugs on the coronary circulation were investigated.Young dogs with open-chest and artificial respiration, ranging in weight from 10 to 15 kg, were used under sodium thiopental (Ravonal) anesthesia. The left coronary artery was perfused from the animal's own right femoral artery and the coronary inflow was measured with rotameter. Then one ml. of drug solution, which contained 1×10-9-10-4 gm epinephrine, 1×10-3-10-4 gm norepinephrine, 1×10-9-10-5 gm acetylcholine, or 1×10-7-10-4 g ATP-Na, were injected into the tubing leading to the coronary artery from the rotameter using 10 seconds, and the changes of coronary blood flow were calcutated.Results were as follows : 1) Doses above 1×10-3-10-7 gm of epinephrine increased coronary flow. In these cases, the coronary flow was increased slowly after the injection and duration of the increase in coronary blood flow was comparatively long. It often lasted several minutes. The increase in coronary blood flow was almost always associated with the elevation of blood pressure and change of heat rate. When large doses of epinephrine was injected, initial decrease in coronary blood flow was followed by a marked increase.2) The effect of norepinephrine was essentially the same as that of epinephrine in quality. Doses above 1×10-7-10-6 gm of norepinephrine increased coronary blood flow, heart rate and blood pressure. Long sustained increase in coronary blood flow was observed. When large doses were injected, initial decrease was also observed.3) Injection of small doses of acetylcholine or ATP-Na often increased the coronary blood flow without remarkable changes of blood pressure or heart rate. Large doses of acetylcholine (1×10-6gm or more) decreased heart rate, blood pressure and coronary blood flow. The effects of acethylcholine and ATP-Na on coronary blood flow appeared more rapidly than those of epinephrine, but lasted for 15-30 seconds.4) As mentioned above, acetylcholine and ATP-Na increased coronary blood flow without remarkable changes of heart rate and blood pressure, but in no experiment epinephrine or norepinephrine augmented the coronary blood flow without any evinence of irritation of myocardium, such as elevation of blood pressure, increase in heart rate and vigor of myocardial contraction. Therefore it can be concluded that in this experiment the direct coronary vasodilatative effects of acetylcholine and ATP-Na were demonstrated, but such effects of epinephrine and norepinephrine were not observed. From present data the presence of the direct action of epinephrine and norepinephrine cannot be decided. But in seems that the direct action of these drugs are vasoconstrictive to coronary artery. At least it appears that the coronary vasodilatative effects of these drugs chiefly result from the hemodynamic and metabolic changes induced by their effects on the myocardium.
  • 第1編 好塩基球数の場合
    荒瀬 進
    医療
    1964年 18 巻 2 号 119-121
    発行日: 1964年
    公開日: 2011/10/19
    ジャーナル フリー
  • 第2編好酸球数の場合
    荒瀬 進
    医療
    1964年 18 巻 2 号 122-124
    発行日: 1964年
    公開日: 2011/10/19
    ジャーナル フリー
  • 第2編 家兎末梢流血中Heinz小体と試験管内形成促進法による核小体との関係について
    松浦 貞章
    岡山医学会雑誌
    1959年 71 巻 10-2 号 6995-7008
    発行日: 1959/09/30
    公開日: 2009/08/24
    ジャーナル フリー
    After giving blood depletion, phenylhydrazine, hydrochloric acid hydroxylamine, carbon tetrachloride, and carbon black to rabbits the author estimated the number of Heinz bodies in the circulating blood of each animal and the number of Heinz bodies appearing in the in vitro accelerating test, and studied the relationship between the two appearances.
    1. In the circulating blood of normal rabbits no Heinz body can be recognized. By the in vitro accelerating method erythrocytes containing Heinz bodies amount ot 66.7 per cent.
    2. In the circulating blood of the rabbits depleted of blood once in a small or a large amount, or successively depleted of blood in an intermediate amount no Heinz body can be found. the circulating blood of the rabbits depleted of its blood once in a large amount a few Heinz bodies can be detected. By the in vitro method Heinz bodies appear in an inverse proportion to the number of erythrocytes, but there seems to be no relation with the number of Heinz bodies in the circulating blood.
    3. When phenylhydrazine is injected, Heinz bodies appear in the circulating blood in various forms according to the dose of the injection. However, by the in vitro method the greater majority of Heinz bodies appeared in the circulating blood are destroyed, and many Heinz bodies observable in this instance are the ones produced anew in vitro.
    4. In the case injected with hydrochloric acid hydrocylamine, Heinz bodies appearing in the circulating blood are more distinct and erythrocytes themselves are more brilliant than those in the case injected with phenylhydrazine. Even by the in vitro accelerating method Heinz bodies appeared in the circulating biood are destroyed in a lesser number than in the case of the phenylhydrazine injection.
    5. In the rabbits administered with carbon tetrachloride Heinz bodies can be recognized in the circualting blood soon after the injection but they are a few in number and small in size. By the in vitro method the blood taken from 10 to 12 days after the injection an intermediate number of Heinz bodies are formed.
    6. In the case injected repeatedly with carbon black Heinz bodies appear in the circulating blood 2 to 4 days after the initiation of the injection, and by the in vitro accelerating method Heinz bodies increase as long as the injection is kept up, but they decrease in number at the cessation of the injection. It seems that the function of the reticuloendothelial system is responsible for the formation of Heinz bodies in the circulating blood while the carbon black injection itself is greatly responsible for the appearance of Heinz bodies by the in vitro method.
    From these, the Heinz body in the circulating blood has the characteristic traits that are different from those of the Heinz body appearing in the in vitro accelerating method. Therefore even the latter is present in vivo in a preparatory state, it is believed that the latter will not immediately be transformed into the in vivo Heinz body.
  • 荒瀬 進, 近藤 恵
    医療
    1960年 14 巻 10 号 873-879
    発行日: 1960年
    公開日: 2011/10/19
    ジャーナル フリー
  • 吉村 正治, 村尾 誠, 武内 重五郎, 小原 常吉, 百瀬 達也, 河目 鐘治, 岡野 正光, 小林 太刀夫
    日本内科学会雑誌
    1953年 41 巻 10 号 592-598
    発行日: 1953/01/10
    公開日: 2011/02/22
    ジャーナル フリー
  • 岡山醫學會雜誌
    1938年 50 巻 1 号 239-240
    発行日: 1938/01/31
    公開日: 2009/07/09
    ジャーナル フリー
  • 村山 達三
    日本傳染病學會雜誌
    1929年 3 巻 4 号 299-313
    発行日: 1929/01/20
    公開日: 2011/11/25
    ジャーナル フリー
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