A female resident of Hokkaido visited a dermatology clinic after developing dermatitis on her both legs. A flea collected in the patient’s home was identified as the human flea, Pulex irritans Linnaeus, 1758. All circumstantial evidence indicates flea bites as the cause of the dermatitis.
トリプシン調節性卵形成阻害因子 (TMOF) は効果的な蚊の幼虫駆除剤だが，非標的生物への潜在的な毒性に関する情報は限られている．これを調べるために，オニテナガエビを12日間，0, 10, 50および100 mg/ LのTMOF溶液に暴露した．尾の水分，粗蛋白質，および肝膵臓グリコーゲンや組織病理学的所見は影響を受けなかったが，生存および生長はTMOFにより濃度依存的に抑制された．TMOFは実験に用いた最低濃度でもトリプシンおよびキモトリプシンの活性を有意に低下させた．(文責：編集事務局)
We report a case of murine typhus in a 4-year-old boy living in northern Greece. Although the illness started with mild symptoms, a maculopapular rash appeared by the end of the first week of illness followed by marked thrombocytopenia. The detection of IgM antibodies against Rickettsia typhi in the patient’s blood and a positive polymerase chain reaction result combined with sequencing confirmed the diagnosis of infection by Rickettsia typhi. Clinicians in northern Greece should be aware of the disease, even in cases presenting with no specific initial symptoms.
Cat scratch disease (CSD) is a syndrome characterized by lymphadenopathy, fever, and skin lesions following a cat scratch or bite. Bartonella henselae is the primary bacterial agent responsible for CSD. In this report, we describe cases with atypical presentation of serologically proven B. henselae neuroretinitis. In this study, 3 patients with neuroretinitis were evaluated. Animal contact histories; results of ocular examinations and systemic investigations; clinical findings; and treatment compliance of the patients were assessed. All patients denied history of contact with cats or other animals, and they did not have CSD findings. Serologic testing via indirect immunofluorescence assay (IFA) was used to diagnose Bartonella neuroretinitis. The IFA test results were positive for all patients. Two patients were treated with antibiotics. Optic disc edema and macular exudates resolved gradually, and at their last follow-up visits, all signs had disappeared. There was no disease recurrence after the completion of treatment. Serious complications were seen in the untreated patient. In conclusion, B. henselae infection should be considered even when there are no systemic signs and symptoms of CSD in patients with neuroretinitis.