Within-tree variations, whole-tree values and the representative heights for the whole tree value of basic density, and contents of wood components per volume were analysed for two trees of Eucalyptus camaldulensis and

E

. globulus respectively, grown at the seed orchard in Western Australia to do quality breeding.
Within-tree variation of

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. camaldulensis was rather uniform as 500±40, 532 ±34 kg/, but that of

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. globulus was large as 595 ± 69, 616 ± 79 kg/ and the basic density was higher in bark side and upper part in the trunk, to be expected to have higher pulp productivity.
The whole-tree values of

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. camaldulensis were 501, 520 kg/ and those of

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. globulus were 594, 640 kg/ and the difference of the species was observed.
Representative heights for the whole-tree contents ± % were .32. m on

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. camaldulensis, and . and 2. m on

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. globulus regardless the differences in the whole-tree height and the within-tree variation when they were chosen below . m height for sampling an increment core.
Representative heights were also determined for contents of wood components per volume as follows ; for

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. camaldulensis, holocellulose : 1., 2. m, cellulose : 2.. m, hemicellulose : .82. m, lignin : .., 2. m, extractives : 2. m, alkali-extractives : 1. m, and total-extractives : 2.32. m, for

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. globulus, 2. m, 2.32. m, 2. m, .30. m, 2.. m, 2.. m and 2. m in the same manner.
It is expected that whole-tree pulp properties including pulp productivity are predicted by the increment core taken from these representative heights.

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The plasma concentration and urinary excretion of 15 (R)-15-methylprostaglandin (Arbaprostil, CU-) and its 15 (S)-15-methyl epimer (CU- (S)) were studied in healthymale volunteers after a single oral administration of CU-.
Maximum plasma concentrations of CU- following the administration at doses of 20, 40 and 60μg were 127., 152. and 164.2pg/ml, respectively, from . to .hr. On theother hand, maximum plasma concentration of CU- (S) following the administration was. pg/ml at 1.1hr in the case of 20μg dose, 17.pg/ml at 1. hr in 40μg and 44.1pg/mlat 1.hr in 60μg.
Urinary excretion half-lives of CU- and CU- (S) were 1.2-1. hr and . 2-.2hr, respectively. Of the administered dose, 1. 45-1. 65% as CU- and .-1.62% as CU- (S) were excreted in urine until 24 hr after administration.

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Seven hundred fifty mg of cholesterol were fed daily to 32 patients of the ischemic heart disease (IHD) for 2 weeks. Cholesterol amounts in VLDL, LDL, HDL, HDL₂ and were estimated on the th, th and 14th days of cholesterol load. Plasma apoprotein A-I, A-II, , C-II, levels were also measured in 16 from 32 IHD subjects. Apoprotein levels were estimated by a method of single radial immunodiffusion (SRID). Before cholesterol administration, cholesterol amounts in VLDL, LDL, HDL, HDL₂ and were 15.±.mg/dl (mean±SD), 150.±58.1mg/dl 43.±.mg/dl, 16.±.mg/dl and 24.±.mg/dl respectively. Plasma apoprotein A-I, A-II, , C-II and levels were .±. mg/dl, 15.1±.mg/dl, 93.±36.mg/dl, .±1.00mg/dl and .29±1.36mg/dl respectively. After 2 weeks' cholesterol feeding, all plasma lipoprotein cholesterol and apoprotein levels did not change significantly. Correlation coefficients between plasma apoprotein and lipoprotein cholesterol levels were calculated. Apoprotein A-I and A-II correlated with HDL-C (r=.348), HDL₂-C (r=.612) and (r=.569). Apoprotein correlated with total cholesterol (TC) (r=.610), VLDL-C (r=.341), LDL-C (r=.726), HDL-C (r=.432) and HDL₂-C (r=.465). Apoprotein C-II correlated with TC (r=.765), TG (r=.679), VLDL-C (r=.651), LDL-C (r=.461) and HDL₂-C (r=.326). Apoprotein correlated with TC (r=.580), TG (r=.575) and VLDL-C (r=.666). Interapoprotein's correlations were also calculated. Apoprotein A-I correlated with apoprotein A-II (r=.468). Apoprotein correlated with apoprotein C-II (r=.393). Apoprotein C-II correlated with apoprotein (r=.393) and (r=.549). Apoprotein only correlated with apoprotein C-II (r=.549).

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