Abstract
agal afferents on laryngeal respiratory function were investigated with special regard to the effect of stretch and pressure afferents on phasic abductor activity.
Forced inflation of the lung reflexly abolished the activity of the posterior cricoarytenoid (PCA) muscle during inspiration. Forced deflation of the lung resulted in increased PCA activity.
After bilateral distol vagotomy, however, neither inflation nor deflation seemed to affect the PCA activity. In addition, continuous low frequencies of vagal stimulation caused reduction of the PCA activity. On the other hand, continuous high frequencies of vagal stimulation caused increased PCA activity.
The results of the experiments reported upon were discussed in regard to respiratory control mechanism.
