A STUDY ON MECHANISM OF RENAL HYPERTENSION CAUSED BY THE MOVABLE KIDNEY

Abstract

Generally, the view is held that renal hypertension caused by movable kidney in excess of renin seretion results not only from reduction of renal perfusion pressure which is due to twisting, angulation and elongation of the renal pedicle but also from urinary stasis and stimulation of the renal nerves. The author investigated the relationship between the degree and blood pressure in 107 patiens with the movable kidney. Most of them were admitted to our clinic except for 3 outpatients. Moreover, renin measurements were carried out on blood plasma specimens obtained from both renal veins, peripheral vessels and inferior vena cava in 56 patients under supine posture, and on the samples from peripheral vessels in 33 patients after 4 hours' upright posture. The following results were obtained.
1) These patients were classified into 3 subgroups such as 69 normotensives, 20 borderline-hyper-tensives and 18 hypertensives. In ten (55.5 percent) of the hypertensive group the movable kidney was regarded as the etiologic factor and also in nine (45.0 percent) of the borderline-hypertensive group as the same. Additionally, even nine normotensive cases (13.0 percent) seemed to be suspected for the latent mechanism of renal hypertension.
2) Angulation or twisting of the renal pedicle may be the most possible cause of renal hypertension resulting from ischemia in these patients with the movable kidney.