Abstract
Tobacco EIN3 like (TEIL) is a transcriptional factor which binds the promoter sequence of acidic tobacco PR1 gene, sharing 60% identity to EIN3 from Arabidopsis. TEIL exhibited transcriptional activation by binding the specific sequence tebs (TEIL binding sites) (Kosugi and Ohashi, 2000). TEIL was constitutively expressed in all tobacco tissues examined. TEIL transcript was accumulated after wounding preceding basic PR gene in leaves, and expression patterns of TEIL family genes were different from that of TEIL. The analysis of TEIL-overexpressed and TEIL-suppressed transgenic tobacco plants indicated that TEIL was positive regulator of wound-signaling involved in basic PR gene expression. Further it was suggested that TEIL was related to leaf senescence, triple-response, and shaping of flower. Here we found that the profile of wound-induced ethylene emission in TEIL-suppressed tobacco was different from that of Wt tobacco, and the sensitivity to fungal pathogen R.solani was enhanced in TEIL-suppressed tobacco.
