Abstract
Systemic acquired resistance (SAR), one of plant defense systems, is induced by pathogen infection through salicylic acid (SA) accumulation. SAR has been well characterized and set of pathogenesis-related (PR) genes has been identified as SAR marker genes. The induction of disease resistance by high temperature treatment has been reported in cucumber, in which SA is likely to take part. To investigate the detailed mechanism of this resistance, effects of high temperature treatment in Arabidopsis was examined. The treatment induced the expression of PR genes, the accumulation of SA, and disease resistance. Analysis using the SA-biosynthesis mutant sid2 revealed that high temparature-induced disease resistance required SA biosynthesis.