Abstract
Since the discovery of Scheinker (1945)9c) the cerebral hemorrhage in the apoplexy is believed by many pathologists to originate from the rupture of altered veins showing special and predominant localization and accompanied by special alteration of the cerebral vein and artery, including hyalinous degeneration of cerebral arterioles, and of the nerve parenchyma.
In the authors' experiments the first attempt to produce experimentally a cerebral hemorrhage with such conditions showing a superficial but close resemblance to human cerebral arteriosclerosis was successful in all rabbits treated by the authors' original procedures using high molecular weight substance as an atherogenic substance and vasoactive hormones as accelerator and showing a fatless type of atheroma which has never been produced until today.
