Abstract
The names, Anisakis larva type I and Terranova type A, have been changed to Anisakis simplex larva and Pseudoterranova decipiens larva respectively. A disease caused by the former larva has been called anisakiasis. Ipropose a term pseudoterranovasis for the disease caused by the latter larva. The total number of reported cases of anisakiasis and terranovasis in Japan has reached 11, 777 by May, 1988. Recent changes in migration of paratenic host near Japan seas have altered frequency of infected fish, resulting in increases of pseudoterranovasis in the northern parts of Japan and anisakiasis in the southern. Accuracy in making a diagnosis of intestinal anisakiasis should further be intensified. The fact that a patient ate paratenic host fish, in combinaiton with several clinical findings, makes a diagnosis of anisakiasis or pseudoterranovasis almost certain. But, in order to make a definite diagnosis of the disease, a worm should be identified by seroimmunological examination using monoclonal antibody of the disease. Some characteristic difference of two larvae, and obstacle components of larvae for human tissue have been districted in this paper. In a development of preventive law in anisakiasis or pseudoterranovasis, the Japanese should learn the Holland's law in forcing the fishery to freeze all fish. Facing a strong resistance by Japanese fishery and retailers, to make such a law in this country is not straihgtforward. We the Japanese medical researchers, therefore, should seek an alternative, preventive way.
