2002 Volume 45 Issue 5 Pages 329-333
A 28-year-old woman reporting polydipsia, polyuria, and weight loss was diagnosed with type 2 diabetes by her physician, and underwent insulin treatment. She was admitted to our hospital due to paresthesia in both lower extremities with rapid improvement of glycemic control. Neurological examination showed sensory abnormality in both lower extremities. Nerve conduction studies showed decreased tibial motor nerve conduction velocity, increased tibial F-wave minimum latency, and decreased sural sensory nerve action potential. She did not have diabetic retinopathy or nephropathy. Paresthesia and abnormalities of nerve conduction studies improved with time, and paresthesia disappeared 6 months later. Her clinical course was thought to be compatible with post-treatment neuropathy.
