Abstract
In Report II the author suggested that an important trigger mechanism of the adrenalin pulmonary edema was a relative increase in the venous return (over the left ventricular output) and that the mobilization of depot-blood was a cause of this increase in the venous return. In this part of study the author analysed the role of the autonomic nervous background of such an increase in venous return. It was suggested that a basic trigger mechanism of the adrenalin pulmonary edema was an abrupt increase in the venous return due to the mobilized depot blood flow, followed by an abrupt cessation of the blood circulation by arteriolar contraction. It appeared that such changes in the venous return were sympathetic in origin and that the parasympathetic nerve activity tended to slow the rate of the venous return.
