On the cellular basis of reperfusion arrhythmias in rabbit ventricular myocytes rabbit ventricular myocytes : Induction of Ca2+ overload and triggered activity by reactive oxygen intermediates
Author's Organization:Department of Physiology, Saga Medical School Department of Physiology, Saga Medical School Department of Physiology, Saga Medical School Department of Physiology, Saga Medical School The Rayne Institute, St. Thomas' Hospital