Acute Kidney Injury by Renal Hemosiderosis Secondary to Primary Cold Agglutinin Disease Associated with an Excessive Alcohol Intake

Aya Imafuku; Go Yamamoto; Koji Takemura; Eiko Hasegawa; Naoki Sawa; Masahiro Kawada; Akinari Sekine; Junichi Hoshino; Kenmei Takaichi; Takeshi Fujii; Kenichi Ohashi; Yoshifumi Ubara

doi:10.2169/internalmedicine.0710-17

This article has now been updated. Please use the final version.

Acute Kidney Injury by Renal Hemosiderosis Secondary to Primary Cold Agglutinin Disease Associated with an Excessive Alcohol Intake

Aya Imafuku, Go Yamamoto, Koji Takemura, Eiko Hasegawa, Naoki Sawa, Masahiro Kawada, Akinari Sekine, Junichi Hoshino, Kenmei Takaichi, Takeshi Fujii, Kenichi Ohashi, Yoshifumi Ubara

Author information

Keywords: acute kidney injury, renal hemosiderosis, intravascular hemolysis, cold agglutinin disease, lymphoplasmacytic lymphoma, excessive alcohol intake

JOURNAL OPEN ACCESS Advance online publication

Article ID: 0710-17

DOI https://doi.org/10.2169/internalmedicine.0710-17

The final version of this article is now available: Vol. 57 (2018), No. 22 pp. 3261-3265

Details

Abstract

Renal hemosiderosis occurs in the context of severe intravascular hemolysis, with the most common cause being paroxysmal nocturnal hematuria. Patients with cold agglutinin disease (CAD) have relatively mild hemolysis, and acute kidney injury (AKI) due to renal hemosiderosis has not been reported. We encountered a patient with CAD caused by lymphoplasmacytic lymphoma who developed AKI secondary to renal hemosiderosis after an excessive alcohol intake.

Corresponding author

Register with J-STAGE for free!