Abstract
The distal lenticulostriate arteries, the predilect site of cerebrla hemorrhage, show ultrastructural damage of the media in hypertensive patients. The proximal segments of the same artery and the cortical circumflex arteries are normal. Thus such medial damage may be important in pathogenesis of arterial rupture and massive hemorrhage in patient with hypertension.
Vasomotor innervation was significantly dense in the lenticulostriate arteries and circle of Willis than in the cortical circumflex arteries. Such rich vasomotor supplies may explain part of pathogenesis of hypertensive medial damage predisposing to rupture at the bifurcation.
In patients with rest angina the coronary arteries, proximal three main branches were often involved by small cell infiltration at the adventitia or the surrounding including vasomotor nerves regardless intimal thickening. The patients show a fresh coronary thrombosis caused fetal myocardial infarction. The repeated coronary spasm led to myocardial infarction may conducted through the vasomotor nerves which were in easely stimulative state by inflammatory cell infiltration.
