Abstract
Aspirin, kanamycin and acoustical stimulation were examined as a possible causes of reversible or transient tinnitus in a retrospective study of case reports. Early observations of cochlear injury assessed morphologically or electrophysiologically were considered in an effort to reconcile the clinical features and experimental observations concerning the origin of cochlear tinnitus. The mechanism of tinnitus is not known, but a clarification of the aspects involved are considered here.
