抄録
1. In the dog anesthetized with pentobarbital sodium the unilateral ureter was extraperitoneally exposed and to three localized regions in the upper, middle and lower parts were applied the plethysmographic tambours which enabled to record the ureteral motility without disturbing the flow of urine.
2. It is often noted that the peristalses recur at a fairly rapid succession (period: 3.4±0.9 sec.) and in uniform amplitude.
3. When the peristalses are produced at an exceedingly rapid succession (period: 2.4±0.3 sec.) in the pace maker region, every other peristalsis is reduced in strength or abolished in the lower part.
4. The electric stimulation of the extraureteral sympathetic as well as parasympathetic nerves always produces on the ureter the increase of tone accompanied with the acceleration of the rate of peristalses. However, the regional difference is noted as to the degree of excitation. a) As to the rate of the peristalses the pronounced accelerating effect is produced by stimulating the splanchnic nerve, and the vagal, hypogastric and pelvic nerve show the effect much less than that obtained by splanchnic stimulation. b) As to the muscle tone the splanchnic nerve exerts in particular the remarkable influence on the upper part of the ureter, while the hypogastric as well as the pelvic nerve has the influence especially on the lower part, and the effect of the vagus nerve is of only a slight degree through the whole length of the ureter.
5. Both adrenaline and acetylcholine exert on the ureteral motility the excitatory actions essentially similar to those obtained by stimulating the extraureteral sympathetic as well as parasympathetic nerves.
6. Both atropine and hexamethonium depress the ureteral motility. Morphine exerts the excitatory action on the ureteral motility.
7. From the results described in 4 and 5 it may be concluded that both sympathetic and parasympathetic nerves lack opposing action on the ureteral motility.
