Abstract
We have recently proposed that LATE ELONGATED HYPOCOTYL (LHY) and CIRCADIAN CLOCK ASSOCIATED 1 (CCA1) are essential components for circadian clock function and flowering time regulation in Arabidopsis. Over-expression of clock-associated genes such as LHY, CCA1 and LKP2 causes a dominant late flowering phenotype under LD. To isolate novel mutations which closely associated with clock function, we are screening Arabidopsis mutants similar to the gain-of function mutants of LHY, CCA1 and LKP2. Recently we have isolated such a mutant from one of our T-DNA tagged transgenic populations. We named it dll1 (dominant late flowering under long day 1). Consistent with the late flowering phenotype, expression level of a floral activator gene, FT, is significantly lowered in this mutant under LD. Details on genetic interactions between the dll1 and other flowering mutations will be reported.
