Abstract
The Arabidopsis mutant cad1(constitutively activated cell death 1)shows a phenotype that mimics hyper-sensitive response (HR)-like cell death. The mutant was observed the significant increase of salicylic acid (SA) levels and activated defense mechanism during pathogen infection. We therefore concluded that the CAD1 negatively controls the SA-mediated pathway of programmed cell death in plant immunity. (Plant Cell Physiol. 2005, 46: 902-912)
Induction of systemic acquired resistance (SAR) results in accumulation of SA in both local cells and systemic organs in coincidence with induction of pathogenesis-related (PR) genes as molecular marker for the SAR. To evaluate the relationship between the SAR and CAD1 function, we used DEX-induced RNAi for the CAD1 knockdown in this study. The results will be reported.
