Journal of the Japanese Association for Infectious Diseases
Online ISSN : 1884-5681
Print ISSN : 0021-4817
ISSN-L : 0021-4817
Investigation of Peripheral Blood Vessels of “Ekiri” Patients According to the Values after Landis-method
II Discussions about mixing of peripheral stagnant blood in the circulating blood resulting from the pressure after Landismethod and the phenomena supporting this conception
Hiroshi WADA
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1959 Volume 33 Issue 4 Pages 324-331

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Abstract

In Report II, a new interpretation of Landis method was attempted by the author. Infants completely recovered from dysentery were taken as contrast and the shift of Ht and Gp-I were measured calculated 5, 15, and 30 minutes after 60mm Hg and 80 mm Hg pressure. A simple concentration and dilution range (rectangular hyperbolic range) was added to the related table of Ht and Gp-I shifts. The mechanism of the mixing of peripheral stagnant blood, probably altered already before the pressure, in the circulating blood consequent on the application of pressure was considered as primary approximation against the variation of blood before and after he pressure and a detailed reexamination of the values obtained was carried out.
1) The divergence of erythrocyte and plasma component 5, 15 and 30 minutes. fter 60 mm Hg and 80 mm Hg pressure tended toward concentration as compared with those of circulating blood. As for erythrocyte component, it was found in the range of greater concentration than plasma component (A range called by the author).
2) The divergence of the values was after 60 mm Hg pressure as well as after 0 mm Hg pressure obviously greater in 15 minutes than in 5 minutes, while it tended again toward the original point in 30 minutes.
These results could not be xplained by Landis' conception, but easily by ours, i. e. “mixing of peripheral sta gnant blood in the circulating blood consequent on the application of ressure”.
These data were thought to result from the greater forcing-out effect of 80 mm Hg than 60 mm Hg pressure. The greater divergence 30 minutes after 80 mm Hg than 30 minutes after 60 mm Hg pressure might be reduced upon a possible little residue of peripheral blood in the former without having been forced out in the circulating blood, because the amount of circulating blood decreases in the rise of pressure inflicted.
Consequently the conception, “mixing of the peripheral stagnant blood in the circulating blood”, might well explain the above mentioned phenomena. We might conclude that the blood examination before and after the pressure would contribute much to the investigation not only of the peripheral stagnant blood but also the peripheral blood vessel themselves.

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