Abstract
Using normal rabbits, solutions of FMN and FAD and percolations of kidney and liver were added each to the blood and to the homogenate of kidneys, liver and small intestines. Riboflavin was measured after the incubation for 30 minutes at 37℃. On the other hand, blood was hourly taken from perpheral veins, renal arteries and renal veins of normal dogs sujected to intravenous injection of 5mg of riboflavin, FMN and FAD each and the riboflavin value in the blood was measured. In kidneys and blood the dephosphorylation of ribflavin was observed, but not in liver and small intestines. The kidney seems to play a very important role in the dephosphorylation of riboflavin.
