Japanese Journal of Electrocardiology
Online ISSN : 1884-2437
Print ISSN : 0285-1660
ISSN-L : 0285-1660
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Volume 31, Issue 1
Displaying 1-13 of 13 articles from this issue
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Editorial
  • A Novel Lamin A/C Gene Mutation in Siblings Treated with Cardiac Pacemaker
    Kan Sano, Eiichi Watanabe, Takeru Makiyama, Tatsushi Uchiyama, Yoshihi ...
    2011 Volume 31 Issue 1 Pages 18-24
    Published: 2011
    Released on J-STAGE: August 02, 2011
    DOIhttps://doi.org/10.5105/jse.31.18
    JOURNAL FREE ACCESS
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    Lamins belong to the intermediate filament gene super-family, which is the main architectural component of the inner nuclear membrane, and influences gene duplication and expression. Lamin A/C gene (LMNA) mutations cause Emery-Dreifuss muscular dystrophy, which is characterized by a triad including joint contractures, muscle weakness, and abnormalities of the conduction-system, and cardiomyopathy. LMNA has also been detected in patients with progressive conduction-system disease and cardiac dysfunction but without muscular dystrophy, which is called cardiolaminopathy. The majority of cardiolaminopathy patients die due to heart failure or ventricular tachyarrhythmias. We report 3 cases (the average age, 49.6 year-old at the time of the implantation of the cardiac pacemaker, male/female=1/2) with a novel nonsense mutation (Q258X) that received cardiac pacemakers for bradycardia.
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  • A Case Report of Successful Resuscitation Following Ventricular Fibrillation Secondary to Wolff-Parkinson-White Syndrome
    Shinya Sugiura, Eitaro Fujii, Michiharu Senga, Takahiro Yonekawa, Hiro ...
    2011 Volume 31 Issue 1 Pages 25-33
    Published: 2011
    Released on J-STAGE: August 02, 2011
    DOIhttps://doi.org/10.5105/jse.31.25
    JOURNAL FREE ACCESS
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    We present a case of a 14-year-old male with Wolff-Parkinson-White (WPW) Syndrome, who was successfully resuscitated from ventricular fibrillation which occurred during basketball practice. He had no family history of unexplained sudden death or syncope. Bystander-initiated cardiopulmonary resuscitation (CPR) was provided there, and shocks using an automated external defibrillator (AED) were delivered by the rescue crews. Fortunately, the patient recovered without after-effects. An electrophysiological study was performed; atrioventricular reciprocating tachycardia and fast-slow type of atrioventricular nodal reentrant tachycardia were induced. Successful catheter ablation of the accessory pathway and slow pathway was performed.
    It is unusual that cardiac arrest is the first symptomatic manifestation of WPW syndrome. This case supports the concept of indication for management of asymptomatic patients with WPW syndrome including electrophysiological study and catheter ablation because the catheter ablation of the accessory pathway is still very effective and low risk.
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  • Magnetocardiographic Spatiotemporal Analysis of Intraventricular Conduction Delay in Patients with Bundle Branch Block
    Kuniomi Ogata, Hiroshi Takaki, Akihiko Kandori, Tsuyoshi Miyashita, Em ...
    2011 Volume 31 Issue 1 Pages 34-44
    Published: 2011
    Released on J-STAGE: August 02, 2011
    DOIhttps://doi.org/10.5105/jse.31.34
    JOURNAL FREE ACCESS
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    Since magnetocardiographic signals (MCG) rapidly decay with the distance between the sensor and the electrical source, the conventional recording method over the anterior chest may not provide sufficient information to evaluate the whole heart’s electrical activation. We thus recorded MCGs over both the anterior and posterior chest walls to perform spatiotemporal analysis of intraventricular conduction delay due to complete left and right bundle branch block (CLBBB and CRBBB, respectively) . The end of QRS was semi-automatically determined on anterior and posterior MCGs. In CLBBB, the QRS lasted longer (14±8 msec) in posterior MCGs than in anterior MCGs. In contrast, subjects with CRBBB showed longer QRS in anterior MCGs than that in posterior MCGs. The pseudo-electrical current mapping revealed that the latest activation site in CLBBB was on the left central area of the posterior MCGs (corresponding to the posterior wall of the left ventricle) , while that in CRBBB was on the right central to inferior area of the anterior MCGs (corresponding to the free wall of the right ventricle) . Our data indicated that analysis of anterior and posterior MCGs is useful for quantifying the intraventricular conduction delay due to bundle branch block and localizing the site of conduction delay.
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