Abstract
1) Ackermann and Kurz's report about the growth of Influenza virus in Ehrlich ascites tumor was confirmed by our stock strain of Ehrlich ascites tumor and Ackermann's strain of Influenza virus. In addition the process of oncolysis was examined cytochemically and cytologically. It was also noted that the ratio of infectivity to hemagglutination titer of infected ascites fluid was low.
2) A new host virus system, AsWS influenza virus and Yoshida ascites tumor system was reported. In this system, the growth pattern of the virus was different from that in the system of Ehrlich ascites tumor in two points, a) hemagglutination was observed only in the emulsion of cell sediment of infected ascites fluid and not in supernatant., b) growth curve showed a peak at 2 or 3 days after infection.
3) The process of oncolysis of Yoshida ascites hepatoma by the virus was examined cytologically and cytochemically.
4) Relationship between the capacity to support viral propagation and canceriyation of liver cells by DAB was discussed.
The anthers wish to express their appreciationto Dr. K. Ogasawara and Dr. S. Hibino for their helpful criticism.
