Abstract
Electron microscopy revealed that panarteritis in the mesenteric arteries of spontaneously hypertensive rats treated with high-cholesterol diet-feeding and vitamin D2-loading might be produced by fibrin insudation and leukocytic infiltration in some segments of the arteries which showed thickening of wall due to marked proliferation of modified smooth muscle cells with partial cytoplasmic degeneration in response to the vascular damage by hypercalcemia.
