抄録
The etiologies of congenital cerebral palsy are varied and very complicated. Medical evidence shows that anoxic brain damage accounts for a very high proportion of all cases of cerebral palsy.
During the years 1963 to 1970, 1088 cases with cerebral palsy between 1 and 9 years were admitted to Kyushu University Hospital.
376 cases (34.3%) gave a history of neonatal asphyxia.
160 cases (14.7%) gave a history of severe neonatal jaundice, which was complicated further by premature birth in 52 cases.
283 cases (26.3%) were premature at birth.
The patients with cengenital malformations which did not appear to be directly related to the abnormalities of the nervous system were found in 43 cases (12.8%), in contrast to 3% in the control group.
There was evidence in some cases of an abnormal family history, suggesting a genetic factor. Areliable history of cerebral palsy occurring in sibs was obtained in 9 families. The risk to subsequentsib of congenital cerebral palsy was 17 per 1, 000. On the other hand, there was a raised frequency of parental consanguinity in the cases with no history of birth abnormality (11.9% in contrast to 5-6% in the control group).
Prenatal, perinatal and postnatal cares are needed equally in order to prevent cerebral palsy.
