The purpose of this study was to elucidate the effectiveness of the experimental training session, which consisted of 8-weeks leg strength endurance training, 8-weeks detraining, and 8-weeks retraining, in a stroke patient with hemiparesis. Three sets of the leg strength endurance training which was composed of consecutive 50 knee extension at the angular velocity of 180° /sec were performed three times a week. The results obtained were: 1) Muscle endurance in both affected and unaffected sides was improved after the session; particularly, the improvement tended to be comparatively prominent in the 4th weeks of the training. 2) During 8-weeks detraining, the muscle strength of unaffected side was maintained, whereas that of affected side showed remarkable deterioration. 3) It was found that the subject kept a very high level of gait amount during detraining, which implies that the walking in which the unaffected side was used dominantly may contribute to keeping the muscle strength to some extent. 4) During 8-weeks retraining, led the temporarily decreased muscle strength of both affected and unaffected sides to some improvement. In 50 trials of muscle out put, the measured values of peak torque of affected muscles became constant after thetraining. It follows from what has been elucidated that muscle endurance of lower extremities of the stroke patient with hemiparesis was improved by the training of relatively high speed. It was further suggested that the neurotic control can possibly be improved by retraining.

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This study was designed to elucidate the way of half kneeling of hemiplegic patients after stroke. Subjects consisted of 69 hemiplegic patients after stroke, all of whom can kneel up independently though in need of watching, and the number of subjects who first put forward the unaffected leg and that forward the affected leg was compared. According to the results obtained, the number of subjects who first put forward the unaffected leg was significantly more than that forward the affected leg, regardless of the elapsed time from stroke and the affected side. It was also revealed that the former was significantly more than the latter among subjects of Br stages III and VI. However, subjects of Br stages of IV and V showed no significant difference between the numbers.

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The characteristics and difference in fatigability after driving were analyzed both in the disabled and healthy persons from the kinesiological and medical viewpoints: former was composed of the reaction time, flicker perimetry, stationary and moving visual acuity, grasping power, etc., and the latter of hemanalysis, blood pressure, etc. It was found from the results obtained that the healthy persons became kinesiologically more fatigued after driving compared with the disabled persons, and that no fatigability was observed in both of them by medical examinations.

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The purpose of this study was to examine how circulatory responses are influenced by changes in angular velocity during isokinetic trunk rotatory exercise.Eight healthy adult males performed exercises at three different angular velocities, 60° /s, 90° /s, and 120° /s, for 20 sec each, and their heart rate (HR), systolic blood pressure (SBP), diastolic blood pressure (DBP), and mean blood pressure (MBP) were measured. It was found that DBP was significant higher during the exercise at the angular velocity of 120° /s than during that of 60° /s and that SBP was also significantly higher during that of 120° /s than those of 60° /s and 90° /s. There was a significant difference in recovery time for HR between the exercises at the angular velocities of 60° /s and 90° /s, and 60° /s and 120° /s. Recovery time for both DBP and MBP was significantly longer after the exercise of 120° /s than after that of 60° /s. These results suggest that the exercise at the angular velocity of 60° /s may be the optimum exercise, putting the least burden on the circulatory system. However, in case the exercise is applied to patients with circulatory diseases, careful consideration will be necessary for the length of exercise period.

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Our concern in this study was to examine the relationship between the electrocardiographic findings and cerebral lesions and biochemical profiles in 227 patients with chronic cerebrovascular disease. For the patients studies, 132 had cerebral infarction, 45 had cerebral hemorrhage, 4 had cerebral thrombosis, 11 had subarachnoid hemorrhage, and 35 had the second attack. It was elucidated that 78.4% of the patients showed abnormal electrocardiographic waves;89.5% of those with subarachnoid hemorrhage, 86.1% with cerebral hemorrhage, 84.3% with cerebral infarction, and 57.1 % with cerebral thrombosis. Abnormal electrocardiographic waves frequently observed consisted of myocardiopathy, supraventricular extrasystole, myocardial infarction, and left axis deviation, being not influenced by lesions. Approximately 80% of the patients over 60 showed abnormal electrocardiographic waves, and the incidence tended to rise with increasing ages. In addition, 86.2% of the patients with a complication of hypertension and 81.8% of diabetes mellitus were found to indicate abnormal electrocardiographic waves. These results made it clear that there was a high incidence of abnormal electrocardiographic waves in the patients with chronic cerebrovascular disease. This suggested that we should check the electrocardiographic findings of those patients when performing therapeutic exercise.

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In order to examine the relationship between muscle volume and anaerobic threshold (AT), we measured ventilatory threshold(VT), an estimated value of AT, of four adult subjects during four modes of exercises (flexion-to-extension exercises of right elbow, both elbows, right knee, and both knees). Mobilized muscle volume was estimated by the change in circumference of the muscle mainly used during each exercise.
It was found that the more the muscle volume is mobilized the higher VT becomes, suggesting that different VT value obtained during exercises of upper extremity ergometer, lower extremity ergometer, or treadmill may result from a difference in muscle volume mobilized.

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Leg length discrepancies are caused by inclinatio pelvis and scoliosis as well as joint deformity like shortening in bone length or hip joint dilocation. They are considered to play a causative role in mechanically inducing poor posture, leading the upright posture to be flexed bilaterally. For the past two and half years, we measured leg length discrepancies in a supine position and torsional inclination of pelvis and hip in a prone position of a total of 809 patients who had admitted to our hospital. In this study, the incidence of leg length discrepancies was examined and the causative factors were analyzed. It was found that the incidence was extremely high, and the shortening in right leg was especially prominent. With increasing ages, the shortening in left leg was found to become prominent, and that in both legs was also observed in some cases. Leg length discrepancies were found to be caused mainly by architectural changes such as muscle or joint contracture in pelvis or lower extremities and subluxation, rather than morphological changes. This study also introduced the analysis of dorsal posture in a prone position.

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In a 34-year-old female patient with insulin dependent diabetes mellitus (IDDM), who was further diagnosed as brittle diabetes, the effect of therapeutic exercise at the exercise intensity of 40% V02max was examined. After 3-week exercise, blood glucose value at starvation and insulin uptake were significantly decreased (P<.05) and the range of daily fluctuation of blood glucose value became smaller due to a combination of the exercise and supplementary food intake after exercise (P<.05). It was further noted that blood glucose value at starvation before breakfast was decreased with the lapse of time. These results indicate that 1) those changes after the exercise resulted from an increase in insulin.sensitivity in periphral tissues, 2) these in the risk of her developing delayed hypoglycemia several weeks after the initiation of the exercise, and 3) continuing control system for hypoglycemia is vital tothe treatment for IDDM patients.

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