A 19-year-old female had been treated with DPH medication, and on the first visit to our clinic, she presented with gaze nystagmus to the right and left side, saccadic ETT and total depressed OKP pattern suggestive of cerebellar dysfunction.
Concentration of DPH in blood reached the toxic level.
By the withdrawal of DPH medication, the concentration of DPH in blood was reduced and the findings of ETT, OKP and gaze nystagmus were improved.
In this case, it was speculated that a reversible lesion due to DPH intoxication existedinot only in the cerebellum, but also in the brain stem.
