Abstract
The present investigations, in which adrenaline was introduced into a vein of a normal dog at the rate, which was chosen to imitate approximately the rate of epinephrine out-put after haemorrhage of about one third or two fifths of the total blood volume, given in previous papers by the present writers, may justify us in concluding that the augmented epinephrine out-put from the suprarenal bodies after haemorrhage plays at least a highly important rôle in causing hyperglycaemia after haemorrhage.
