Background: Robot‐assisted pancreaticoduodenectomy (RPD) enables more precise manipulation and shorter incisions than conventional open pancreaticoduodenectomy (OPD). However, few studies have evaluated the impact of robot‐assisted pancreatic resection on short‐term surgical outcomes and postoperative nutritional status. To address this, we conducted a retrospective analysis of cases from our institution.
Methods: We retrospectively compared short‐term surgical outcomes and postoperative nutritional indicators between patients who underwent RPD and those who underwent OPD at our institution between January 2017 and December 2023.
Results: We included 23 cases in the RPD group and 34 cases in the OPD group. Surgery time was longer in the RPD group than the OPD group (median: 556 minutes vs. 472 minutes), intraoperative blood loss was reduced (111 ml vs. 861 ml), and there were fewer postoperative complications (1 case [4.3％] vs. 12 cases [35.3％]). PNI was 35.3 in the RPD group and 32.5 in the OPD group at 7 days postoperatively; and 37.9 and 35.1, respectively, at 14 days postoperatively, indicating that the RPD group showed earlier recovery of PNI postoperatively.
Conclusion: RPD may contribute to reducing the incidence of postoperative complications and improving nutritional status.

The patient was a 1‐year‐old boy. He was born at 24 weeks and 3 days of gestation with a birth weight of 662 g. At 3 months of age (corrected age: 27 weeks), he underwent extensive small bowel resection and ileostomy for midgut volvulus caused by intestinal malrotation, leaving 20 cm of residual small intestine. The patient experienced prolonged liver dysfunction, jaundice, and renal impairment, as well as poor weight gain and multiple fractures of the extremities. He was transferred to our hospital for long‐term nutritional management.
The patient was 1 year and 3 months old (corrected age: 7 months) at the time of admission and his weight was 3.5 kg. X‐rays showed multiple fractures of the extremities. Laboratory findings showed malnutrition, hypocalcemia, and renal dysfunction (serum parathyroid hormone level, 1174.2 pg/mL). He was diagnosed with secondary hyperparathyroidism associated with malabsorption due to short bowel syndrome and renal impairment.
Post‐admission, the patient was treated for secondary hyperparathyroidism by adjustments to calcium and phosphate supplementation, as well as an increased dose of total parenteral nutrition. After 3 months of hospitalization, his weight increased to 5.71 kg, and his electrolyte imbalances resolved. We initiated a treatment plan that includes continued nutritional management aimed at promoting further weight gain.