抄録
Recently, we experienced 2 cases each of primary acquired and secondary sideroblastic anemias. Among these patients, we examined 55Fe-uptake of erythroblasts in vitro in a case with the primary acquired sideroblastic anemia. In contrast to the cases in aplastic ancmias, this patient showed a similar pattern to that of iron-deficiency anemia; i. e. an increased iron-uptake, besides a shortening of the plasma-iron disappearance curve and a rapid transitional reappearance in the bone marrow. Furthermore, 55Fe-uptake in vitro tends to be decreased by adding vitamin B6, suggesting some attributions of the vitamin B6 to the etiology of the sideroblastic anemia.
