抄録
We have experienced a case of transfusion vivax malaria. A man aged 60, a wrapper, had been in Siberia from October, 1944, to October, 1948, where he suffered jaundice for a month in October, 1946. He had no other trip abroad and no history of malaria in the past. He was in a hospital to treat fracture of the right femoral neck since January 30, 1985, and an orthopedic surgery of the right hip joint was done on June 19, 1985. He was transfused with three packs of red cell concentrates on June 19 and two on the 20th to cover the bleeding at the surgery. He developed moderate fever from July 5 (16 days after the operation), and high fever and nausea and anorexia from July 10, and anemia from July 13. He was diagnosed as Plasmodium vivax malaria on July 17 (28 days after the operation) and treated intravenously with quinine dihydrochloride (400 mg), and then Fansidar (3 tablets for 3 days). He was completely cured by the treatment.
Four of 5 blood donors were Japanese and had no association with malaria. Another donor was an Indian from Punjub State, a known malaria endemic region. After coming to Japan in October 1984, she stayed in Osaka without any malarial symptom. The vivax malaria was most possibly induced by the red cell concentrate from this blood donor. This is the second case of transfusion malaria in Japan since the war-induced transfusion malaria had disappeared in 1965.
Since personnel interchange between Japan and many malaria endemic foreign countries increased heavily in recent years, we have to pay attentions to transfusion malaria in Japan again.
