Relation between Polymorphism of δ-Aminolevulinic Acid Dehydratase and Some Parameters in Lead Workers

Jiusong ZHANG; Masayoshi ICHIBA; Yanping WANG; Shiro YUKITAKE; Katsumaro TOMOKUNI

doi:10.1539/joh.40.77

Jiusong ZHANG, Masayoshi ICHIBA, Yanping WANG, Shiro YUKITAKE, Katsumaro TOMOKUNI

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Keywords: Lead, Pol ymorphism, δ-Aminolevulinic acid dehydratase, δ-Aminolevulinic acid

JOURNAL FREE ACCESS

1998 Volume 40 Issue 1 Pages 77-78

DOI https://doi.org/10.1539/joh.40.77

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Published: 1998 Received: April 09, 1997 Available on J-STAGE: April 07, 2006 Accepted: July 07, 1997 Advance online publication: - Revised: -
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Date of correction: April 07, 2006 Reason for correction: - Correction: CITATION Details: Wrong : 1) Battistuzzi G, Petrucci R, Silvagni L, Urbani FR, Caiola S. &selta;-aminolevulinate dehydrase: a new genetic polymorphism in man. Ann Hum Genet 1981; 45: 223-229.
2) Wetmur JG, Lehnert G, Desnick RJ. The δ-aminolevulinate dehydratase polymorphism: higher blood lead levels in lead workers and environmentally exposed children with the 1-2 and 2-2 isozymes. Environ Res 1991; 56: 109-119.
3) Smith CM, Wang X, Hu H, Kelsey KT. A polymorphism in the δ-aminolevulinic acid dehydrase gene may modify the pharmacokinetics and toxicity of lead. Environ Health Perspect 1995; 103: 248-253.
4) Wetmur JG, Kaya AH, Plewinska M, Desnick R. Molecular characterization of the human δ-aminolevulinate dehydratase 2 (ALAD²) allele: implications for molecular screening of individuals for genetic susceptibility to lead poisoning. Am J Hum Genet 1991; 49: 757-763.
5) Nikkanen J, Hernberg S, Tola S. Modifications of the δ-aminolevulinic acid dehydratase test and their significance for assessing different intensities of lead exposure. Work Environ Health 1972; 9: 46-52.
6) Tomokuni K, Ogata M. Simple method for determination of urinary δ-aminolevulinic acid as an index of lead exposure. Clin Chem 1972; 18: 1534-1536.
7) Morita Y, Sakai T, Tsuruta H, Araki T. Polymorphism of delta-aminolevulinic acid dehydratase and delta-aminolevulinic acid in plasma in lead workers and unexposed subjects. San Ei Shi 1996; 38: S500 (in Japanese).
8) Benkmann HG, Bogdanski P, Goedde HW. Polymorphism of delta-aminolevulinic acid dehydratase in various populations. Hum Hered 1983; 33: 62-64.
9) Hernberg S, Nikkanen J. δ-aminolevulinic acid dehydratase as a measure of lead exposure. Arch Environ Health 1970; 21: 140-145.

Right : 1) Battistuzzi G, Petrucci R, Silvagni L, Urbani FR, Caiola S. δ-aminolevulinate dehydrase: a new genetic polymorphism in man. Ann Hum Genet 1981; 45: 223-229.
2) Wetmur JG, Lehnert G, Desnick RJ. The δ-aminolevulinate dehydratase polymorphism: higher blood lead levels in lead workers and environmentally exposed children with the 1-2 and 2-2 isozymes. Environ Res 1991; 56: 109-119.
3) Smith CM, Wang X, Hu H, Kelsey KT. A polymorphism in the δ-aminolevulinic acid dehydrase gene may modify the pharmacokinetics and toxicity of lead. Environ Health Perspect 1995; 103: 248-253.
4) Wetmur JG, Kaya AH, Plewinska M, Desnick R. Molecular characterization of the human δ-aminolevulinate dehydratase 2 (ALAD²) allele: implications for molecular screening of individuals for genetic susceptibility to lead poisoning. Am J Hum Genet 1991; 49: 757-763.
5) Nikkanen J, Hernberg S, Tola S. Modifications of the δ-aminolevulinic acid dehydratase test and their significance for assessing different intensities of lead exposure. Work Environ Health 1972; 9: 46-52.
6) Tomokuni K, Ogata M. Simple method for determination of urinary δ-aminolevulinic acid as an index of lead exposure. Clin Chem 1972; 18: 1534-1536.
7) Morita Y, Sakai T, Tsuruta H, Araki T. Polymorphism of delta-aminolevulinic acid dehydratase and delta-aminolevulinic acid in plasma in lead workers and unexposed subjects. San Ei Shi 1996; 38: S500 (in Japanese).
8) Benkmann HG, Bogdanski P, Goedde HW. Polymorphism of delta-aminolevulinic acid dehydratase in various populations. Hum Hered 1983; 33: 62-64.
9) Hernberg S, Nikkanen J. δ-aminolevulinic acid dehydratase as a measure of lead exposure. Arch Environ Health 1970; 21: 140-145.

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