1969 年 21 巻 1 号 p. 111-115
The case was 51-years-old male who had deformity of the spinal colum and marked ascites fluid.
At au topsy, main pathological changes were found in the liver. In its cut surface, many yellowish nodules were seen, which were histologically identified as anemic infarct like necrosis of the hepatic cells coexisting with the picture of cirrhosis, although any morphological changes, such as thrombus, embolus or tumor, which may disturb the blood circulation, were found neither in hepatic artery, hepatic vein nor portal vein.
It is generally believed that anemic necrosis of the liver are caused by the occlusion of hepatic artery. In this case, no morphological vidence of the occulsion in hepatic artery could be detected at autopsy.
Therefore, the author assumed a functional occlusion of the hepatic artery in this case; that is, the deformity of the spinal colum, which narrowed the peritoneal cavity and displaced the hepatic artery, might have enhanced the compression and occlusion of the latter by ascites due to liver cirrhosis, followed by the anemic infarct like necrosis of hepatic cells.