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Takeshi Ogura
1989 Volume 12 Issue 5 Pages
438-440
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
441-442
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
443-444
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], Jerzy W. Kupiec-Weglinski, Nicholas L. Tilney
1989 Volume 12 Issue 5 Pages
446-448
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
449-451
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
452-454
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
455-458
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
459-463
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
464-467
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
468-470
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
472-478
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
479-481
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
482-486
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
487-491
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
492-495
Published: 1989
Released on J-STAGE: January 22, 2009
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Shigeo Nomura, Tadashi Kanoh
1989 Volume 12 Issue 5 Pages
496-501
Published: 1989
Released on J-STAGE: January 22, 2009
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We made clinical, immunologic and histochemical studies of 22 cases of AL type and non-AA type pulmonary amyloidosis, none of which were associated with multiple myeloma and other 2 cases of AA type pulmonary amyloidosis, which were associated with systemic lupus erythematosus. Eighteen cases among them had evidence of systemic deposition of amyloid. The majority of our cases were of AL type or of non-AA type. In the former monoclonal proteins were demonstrated in the serum and/or urine by immunofixation. The latter showed amyloid staining with Congo red which was resistant to potassium permanganate, but had no monoclonal proteins in both the serum and urine.
These findings were contrary to the general expectation that chronic pulmonary diseases, such as silicosis, bronchiectasis and lung cancer, are often complicated with AA type amyloidosis.
AL type or non-AA type amyloidosis might occur in association with plasma cell dyscrasia following polyclonal responses to chronic diseases. On the other hand, if inflammatory processes stimulating a production of S-AA, a precursor of AA protein, are more prominent, AA type amyloidosis might occur.
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[in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
502-505
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
506-508
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
509-512
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], David T. Purtilo
1989 Volume 12 Issue 5 Pages
513-514
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
515-518
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
520-521
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
522-524
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
525-530
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
531-536
Published: 1989
Released on J-STAGE: January 22, 2009
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Mikio Kataoka, Yasunari Nakata
1989 Volume 12 Issue 5 Pages
537-543
Published: 1989
Released on J-STAGE: January 22, 2009
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We investigated the functions of peripheral blood monocytes (Mo) and alveolar macrophages (AM) from patients with interstitial lung disease, especially those with sarcoidosis, and normal subjects. AM were obtained by bronchoalveolar lavage (BAL) and Mo from venous blood. We determined the absolute count, chemotaxis, phagocytic index, lysosomal enzyme activity, cytotoxicity, superoxide anion release, IL-1 production and serum and BAL fluid levels of angiotensin converting enzyme (ACE), lysozyme and fibronectin.
In the peripheral blood of sarcoidosis patients, the absolute number of Mo was significantly increased compared with normal subjects, moreover the absolute count of Mo in bone marrow was increased. There was a close correlation between the absolute numbers of Mo in peripheral blood and bone marrow. Chemotaxis and phagocytic index were decreased in patients with sarcoidosis, however superoxide anion production, beta-galactosidase activity and serum levels of ACE and lysozyme were higher in sarcoidosis patients than in normals. There were no differences in cytotoxicity and IL-1 production between sarcoidosis patients and normal subjects.
There was no difference in the number of between sarcoidosis patients and normals. Chemotaxis and phagocytic index, though, unlikely in peripheral blood Mo, were increased significantly in sarcoidosis patients compared with normals. Furthermore, the levels of ACE and fibronectin were significantly higher in sarcoidosis patients than in normals as well. However beta-galactosidase activity was decreased in sarcoidosis patients compared with normals. There was no difference in IL-1 production between sarcoidosis patients and normal subjects.
These data suggest that peripheral blood monocytes arise in the bone marrow and become alveolar macrophages and that both peripheral blood monocytes and alveolar macrophages are activated.
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[in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
546-549
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
550-553
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
554-557
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
558-561
Published: 1989
Released on J-STAGE: January 22, 2009
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Hirofumi Makino, Shuzo Hirakawa
1989 Volume 12 Issue 5 Pages
562-566
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese], [in Japanese], [in Japanese]
1989 Volume 12 Issue 5 Pages
567-569
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
570-573
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
574-576
Published: 1989
Released on J-STAGE: January 22, 2009
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[in Japanese]
1989 Volume 12 Issue 5 Pages
577-580
Published: 1989
Released on J-STAGE: January 22, 2009
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