Minamata disease was officially recognized in May 1956. The disease was caused through the con-sumption of fish and shellfish that were contaminated by a methylmercury compound being discharged from the chemical plant of Chisso Co., Ltd. in Minamata City. Inhabitants living around the Shiranui Sea, who consumed large amounts of the fish, were affected by methylmercury poisoning ; such poisoning was since named Minamata disease, representing its mechanism of occurrence through the food chain.
In 1969, victims and their families instituted a lawsuit against the company responsible, and the court ordered the company to pay compensation for the damage caused by their illegal discharge and lack of ensuring appropriate safety measures. Following the judgment and with public awareness, safety standards for mercury levels in fish and shellfish were set at 0.4ppm of mercury and 0.3ppm of methylmercury.
In 1962, it was demonstrated that methylmercury affects the fetus through the placenta. Although the precise number is unknown, approximately 70 victims have been identified up to the present. The toxic effect of trace amounts of methylmercury accumulated in fish and shellfish on the fetus is being examined on a worldwide scale. Studies conducted in each country have reported that even 10 to 20ppm of methylmercury in a hair sample indicates certain negative effects on the fetus.
The planning of international mercury control regulations was initiated in 2011 to establish an international treaty for mercury control by 2013. Although Japan suggests naming the treaty the “Minamata Treaty”, there are some arguments against such a name in consideration of unresolved problems regarding Minamata disease.
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