THE JOURNAL OF JAPAN SOCIETY FOR CLINICAL ANESTHESIA
Online ISSN : 1349-9149
Print ISSN : 0285-4945
ISSN-L : 0285-4945
Volume 21, Issue 4
Displaying 1-9 of 9 articles from this issue
  • [in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...
    2001 Volume 21 Issue 4 Pages 177-181
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
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  • [in Japanese]
    2001 Volume 21 Issue 4 Pages 182-186
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
    Download PDF (575K)
  • [in Japanese]
    2001 Volume 21 Issue 4 Pages 187-191
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
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  • [in Japanese]
    2001 Volume 21 Issue 4 Pages 192-195
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
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  • Yoshitaka INOUE, Takashi KAWASAKI, Kenichi ABE, Masanori OGATA, Masayo ...
    2001 Volume 21 Issue 4 Pages 196-201
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
    This retrospective study was performed to investigate the changes in plasma ionized calcium (iCa) and magnesium (iMg) concentrations, and their proper supplementation in 16 adult patients whose intraoperative blood loss exceeded 4, 000ml.
    Despite occasional supplementation, iCa concentrations (mmol/l) decreased from 1.06±0.06 to 0.71±0.13 during surgery, and returned toward baseline values by the end of surgery. iMg concentrations (mmol/l) decreased from 0.46±0.06 to 0.19±0.07, and returned to 0.27±0.07 at the end of surgery and to 0.38±0.06 in the first post-operative day without any supplementation.
    A plausible cause of the decreases in plasma iCa and iMg concentrations was hemodilution due to administration of fluid with low concentrations of Ca2+ and Mg2+ to maintain circulation during surgery.
    The volume and electrolyte concentration of fluid administered during surgery must be considered to maintain normal plasma iCa and iMg levels in patients with massive intra-operative bleeding.
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  • Kinya SATO, Mitsuhiko OOHATA, Hiroshi OKADA, Naohisa MORI, Kazuo MURAI ...
    2001 Volume 21 Issue 4 Pages 202-207
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
    Nitrous oxide (N2O) is known to cause an increase in volume and/or pressure in enclosed body cavities as a result of diffusion. In the middle ear cavity, both the middle ear pressure (MEP) and volume increase due to the semicompliant boundaries of this region which include the tympanic membrane and eustachian tube. During general inhalational anesthesia, it seemed that the functions were quickly passive ventilation and comfortable ventilation through the mucosa of the mastoid area. This study examined the relation of the mastoid area and MEP under the start and cessation of N2O.
    During general anesthesia with or without N2O, it seemed that the increase (10.7±2.1mmH2O•min-1) or decrease (2.1±0.5mmH2O•min-1) in MEP without passive ventilation was related to very low circulation in the mastoid area (MEP during N2O administration and the area: r=0.55 (p=0.03); MEP during N2O wash out and the area: r=0.73 (p=0.02)).
    Based on the present results, we observed a positive correlation between the changes of MEP in the mastoid area.
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  • Mikiya OTSUJI, Kanji UCHIDA, Tomoki NISHIYAMA, Mieko CHINZEI, Kazuo HA ...
    2001 Volume 21 Issue 4 Pages 208-212
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
    Surgical correction of congenital portosystemic venous shunt (CPSVS) is sometimes associated with remarkable hemodynamic deterioration and poor outcome due to decreased venous return, portal hypertension and severe splanchnic congestion. This may occur especially in patients with severe hepatic insufficiency. We present the case of a 43-day-old female with CPSVS located between the umbilical portion of the portal vein and the right atrium. Anesthesia was induced with thiopental, fentanyl and vecuronium. In addition to the arterial and central venous pressures, portal venous pressure was monitored intraoperatively. The shunt vessel was ligated after a temporary test occlusion without remarkable hemodynamic alterations. The patient recovered without any complications.
    Intraoperative monitoring of the portal venous pressure along with preoperative evaluation of the liver parenchyma and portal system was useful in the management of CPSVS.
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  • Motoshi KAINUMA, Kyou KAWASE, Morimasa YAMADA, Yoshinobu HATTORI, Tosh ...
    2001 Volume 21 Issue 4 Pages 213-217
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
    Retrograde aortic dissection occurred at the start of cardiopulmonary bypass (CPB) in a patient undergoing aortic valve replacement. This life-threatening complication was strongly suspected due to sudden unexplained hypotension and white discoloration of the aortic root. Transesophageal echocardiography (TEE) immediately established the diagnosis by the emergence of the intimal flap in the ascending and descending aorta. The retrograde aortic dissection extended from the left femoral artery to the aortic root. CPB was interrupted while the cannulation site was changed to the right subclavian artery. CPB was resumed and the closure of the dissection and ascending aortic reconstruction was performed. TEE was particularly useful to guide the surgical repair this complication.
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  • When a Tube Sticks to the Anterior Wall of Trachea
    Takeshi YOKOYAMA, Yukiko KATAOKA, Miho TOMODA, Tetsuya KANBARA, Masano ...
    2001 Volume 21 Issue 4 Pages 218-221
    Published: May 15, 2001
    Released on J-STAGE: December 11, 2008
    JOURNAL FREE ACCESS
    During nasotracheal intubation, sometimes it is difficult to advance a tube just beyond the vocal cord. In such cases, changing the neck position and/or using a Magill forceps could make the tube advance. However, changing the neck position might damage the neck and rough management of the tube might injure tracheal tissue or cause laryngeal edema. We found an easy way to resolve the problem without such troubles.
    When the tube can not be advanced further just beyond the vocal cord, connect the circuit to the tube, close the mouth and another naris and ventilate with 15-20cmH2O two or three times. Subsequently advance the tube again. This procedure is very easy and can advance the tube successfully.
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