As to the intraauricular conduction of the cardiac impulses there are two theories. According to the one, the impulses from sinus node are conducted radially in all directions in the auricles, whereas to the other, only on the specific pathways. In electrocardiograms of a case of partial sinoauricular block, the authors found that the sinus-impulses were not conducted to both auricles, but only to a-v-node, and then further forward to the ventricles on the one hand and simultaneously back-ward to both auricles on the other, bringing about the auricular and ventricular contractions. This finding can be easily understood by the latter theory, and therefore we believe in the existence of the specific pathways from sinus node to a-v-node.

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Rabbits were sensitized with the mixture of diphtheria toxin or anatoxin and rabbit-heart-muscle-phosphatide. After reinjection with the same antigen, their organs, especially heart were investigated histologically. Great degenerative change of myocardium and some pericapillaritis in epicardium were found, and electrocardiogram showed myocardial damage. Without heart-muscle-phosphatide, not so severely changed.

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Although some investigators claim the presence of the vagal cardioaccelerator fibers, there still seems to be a considerable dispute in this problem. While Okinaka and his collaborators confirmed that the cardio-inhibitory fibers of the vagus nerve were localized in the caudal portion of the myelencephalic rootlets of the accessory nerve. Assuming that the probable vagal cardio-accelerator fibers run through the other portion of the rootlets, following experiments were carried out. First only the portion of the rootlets which contain the cardio-inhibitory fibers was cut on the right side intractanially in six dogs. About two weeks thereafter, when the cardio-inhibitory fibers selectively degenerated, cut vagus-stem rootlets of cut upper cervical vagosympathetic trunk were stimulated with faradic current. In five dogs moderate cardiac accelerations were demonstrated. Then in order to heighten the tonus of this vagal cardio-accelerator fibers, the right stellate ganglion was extirpated together with the section of the cardio-inhibitory rootlets on the right side. In this experiment seven dogs were used. In three, cardio-accelerations were found without the use of atropine. In one of them the acceleration was especially marked, before stimulation 4.5 beats per 5 seconds, after stimulation 10 beats per 5 seconds. In two of the remaining four, cardiac accelerations were observed only after the injektion of atropine. In these cases some portions of the rootlets which contain the cardio-inhibitory fibers were found intact at the autopsy. From these results, the presence of efferent cardio-accelerator fibers in the vagus nerve was definitely confirmed.

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2) When ecg. shows the biphasic curve, the thickening of epicardium by increase of connective tissue and cellinfiltration is found in highest degree.

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The changes in R_I-height of guinea pig in each experimental course were investigated. A. Exp. I (Head→Tail×10") The general type of change in R-height in the experiment (3.5, 6, 8g) is the "Enlarged type," and the time indicating the maximal enlargement of R during exposure to the acceleration and the time returning to the original R-height after the cessation of centrifugalization are the same as those of P in the same kind of experiment. (See the report I) Generally the max. enlargement rate (%) is almost similar for all kinds of exposed. B. Exp. II (Tail→Head×10") In most cases the type of change in R-height in the experiment (3.5, 6g) is the "Reduced type," and the times indicating the max. reduction of R, and returning to the original R-height in the experimental course are the same as those of P in the same kind of experiment. In general the max. reduction rate (%) is like in both 3.5g and 6g-experiment. C. Exp. III (to L. & R. along the transverse axis×10") The general type of change in R-height is the "Englarged type" and it's change in experiment (3.5, 6g) is the very same as that seen in Exp. I. Generally the max. enlargement rate (%) is nearly equall in various kinds of this experiment.

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This report based my findings on study of 512 mostly young persons, who were wounded in their heads in battle. Many of the observations were made extending over a long space of time when the head-wounds were having influence on the blood pressure. The studies showed that an increase in blood pressure at the rate of frequency 10.4 percent was one of the characteristic symptoms of the head wounded patients, especially remarkable for many of the war wounded persons suffering from diencephalon and hypophysis. The writer concluded that these experiments indicated strongly that the high blood pressure must be the centrical one caused by the external head injury. The tests were made on so many patients who were afflicted with symptoms of between brain, for example narcolepsy, adiposis, perkinsonism, subcutaneous adipose atrophy on the side, acromegaly, polyglobulism and hyperglycemia, or who complained of vegetative nervous symptoms, such as tachycardia, paroxysmal tachycardia, achilly constitution of hands and feet, acrocyanosis, hyper-hidrosis and frequent attack of slight fever. It ought here to be mentioned that an external injury of head is apt to inflict a wound on brain stem and so has vast influence upon the vasomotor centre of between brain and other vegetative nerve centres.

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In electrocardiograms of a case of ventricular parasystole with simple interference the authors found that after Aschner's eyeball pressure test, though sinus action was completely depressed, parasystolic ventricular complexes were retained without being influenced. From this finding being clear that the impulses from parasystolic center can in itself cause paraystole and can introduce the heart into action, it must be separated distinctly from so-called extrasystole, because to the latter, not only the ectopic center, but also the preceeding impulses which give rise to it are essential.

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