JAPANESE CIRCULATION JOURNAL
Online ISSN : 1347-4839
Print ISSN : 0047-1828
ISSN-L : 0047-1828
Volume 25, Issue 12
Displaying 1-6 of 6 articles from this issue
  • ETSU HASHIDA
    1961 Volume 25 Issue 12 Pages 1235-1256
    Published: December 15, 1961
    Released on J-STAGE: April 14, 2008
    JOURNAL FREE ACCESS
    The significance of late or secondary R waves is studied on ninety subjects with miscellaneous electrocardiographic patterns including the normal by means of intracardiac electrocardiography. In classical right ventricular hypertrophy and complete right bundle branch block, late R waves can be attributed to a delay in activation of the right ventricular free wall. In the other patterns including a part of incomplete right bundle branch block, activation of the left ventricular posterior base constitutes these late R waves.
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  • IKUICHI UEYAMA
    1961 Volume 25 Issue 12 Pages 1257-1271
    Published: December 15, 1961
    Released on J-STAGE: April 14, 2008
    JOURNAL FREE ACCESS
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  • HARUO ENOMOTO
    1961 Volume 25 Issue 12 Pages 1277-1286
    Published: December 15, 1961
    Released on J-STAGE: April 14, 2008
    JOURNAL FREE ACCESS
    In order to study clinically the genetic mechanism of the ventricular strain pattern ECG the author studied the relationship between the ventricular strain pattern ECG and various clinical laboratory tests. Materials used consisted of (a) 60 cases of Hypertension in whom ECG were analysed in details and who included 13 cases having left ventricular strain pattern ECG, (b) 58 cases with left ventricular strain pattern and (c) 32 cases with right ventricular strain pattern. In addition to this also studied were 8 cases with the mitral stenosis having upright T waves in V1 V2 or V3 and 9 cases of congenital heart anomalies having upright T waves in V1, V2 or V3 The result of this study is as follows : (1) As to the age factor, the lartest fraction of the patients with left ventricular strain pattern belonged to the age-stratum of 50-70; the largest fraction of the cases with right ventricular strain pattern belonged to that of 30-50. (2) As to the relative incidence of underlying diseases, the group of left ventricular strain pattern consisted of cases with hypertension (45.5%), cases with valvular diseases (22.4%), and cases with cardiovascular syphilis (15.8%); the incidence of arteriosclerosis with- out hypertension was relatively low. The group of right ventricular strain included cases with mitral valvular diseases (56.2%), cases with congenital heart anomalis (18.7%), those with pulmonary diseases (12.5%) and those of syphilis (12.5%). (3) As to the blood pressure factor, the left ventricular strain pattern did not necessarily correlate with the systolic blood pressure, although it occurred often in cases of hypertension. Neither did it correlate with the diastolic pressure, mean blood pressure or venous pressure. While the right ventricular strain pattern was often associated with somewhat increased right ventricular systolic pressure, (right ventricular) end-diastolic pressure, (right ventricular) mean pressure, mean pulmonary artery pressure and pulmonary "capillary" pressure above normal, the elevation of these pressures above normal was not necessarily associated with the inverted T wave ; that is to say, the right ventricular strain pattern did not bear a direct relationship with these pressures. Elevated above a certain limit, these pressures became closely correlatable with the incidence of the right ventricular strain pattern. In short, the incidence of the ventricular strain pattern ECG bears a certain relationship, but not a direct pathogenetical one, with the systemic or intraventricular pressures.
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  • HARUO ENOMOTO
    1961 Volume 25 Issue 12 Pages 1287-1293
    Published: December 15, 1961
    Released on J-STAGE: April 14, 2008
    JOURNAL FREE ACCESS
    The relationship between the strain pattern ECG and the coronary insufficiency has been studied with various methods, such as by recording ECG by oesophageal leads or with the administration of amyl nitrite, O2 load or exercise load. The results were as follows : (1) Thirteen cases of left ventricular strain pattern and 4 cases of right ventricular strain pattern were subjected to the ECG recording from the oesophagus. Of 13 cases who gave the left ventricular strain pattern in chest leads, 5 cases(38.4%) gave the same ventricular strain pattern at the base and posterior wall of the left ventricle. In those who gave right ventricular strain pattern in chest leads normal T waves were recorded from the base of the left ventricle. (2) Amyl nitrite was administered to 11 cases with left ventricular strain pattern and 2 cases with right ventricular strain pattern in the form of inhalation of 0.25cc (5 drops) and ECG were recorded before, 6 minutes after and 12 minutes after, the inhalation. In the group of left ventricular strain pattern this maneuvre caused the T wave inversion and ST depression to become less pronounced, to some extent, than before in CR5 and CR6 in about 70% of the cases. On the contrary STII was depressed in 55% of the cases. (3) Four cases with left ventricular strain pattern and one case with right ventricular strain pattern were subjected to the O2 load, using a complete closed system, i.e., DaviesGilchrist mask, in dose of 3 liter per minute and for the period of 15 minutes ; ECG were recorded before, 1 minute after, 12 minutes after, and 12 minutes after the cessation of, the O2 load. T waves tended to become high in II, CR5 and CR(; by 0.18-0.01 mV ; ST segment was elevated in all cases in CR6, and in a half of case, in CR2 and II and by 0.06-0.01 mV. Twelve minutes after the cessation of the O2 load, R spike became, low, or tended to become low, ST segment depressed and T wave lowered. QTc was prolonged 12 minutes after the start of O2 load ; its change was not consistent one minutes after, or 12 minutes after the cessation of, O2 load. (4) Six cases with left ventricular strain pattern and 2 cases with right ventricular strain pattern were subjected to Master's exercise test in double dose ; ECG were recorded before, 3 minutes after, 10-12 minutes after, the exercise test. In the group of left ventricular strain pattern the T wave became low or more inverted than before in II, CR2 and CR6 in 70% of the cases each ; on the contrary the T wave in CR5 became less inverted than before in 85 % of the cases. The ST segment became more depressed than before in II, CR6 in 83.66% of the cases each ; changes in CR5 were diverse in direction. The magnitude of these changes was small in all cases. QTc was prolonged in 83% of the cases. (5) It is suggested from the above observations on ECG recorded from the oesophageal leads and with 3 types of loading tests (1) that the ventricular strain pattern ECG is closely related to the coronary insufficiency and (2) that such a coronary insufficiency depends much on the coronary factors and (3) that the observed facts can be beautifully explained by Prof. Maekawa's theory of "latent coronary insufficiency". (6) The author discussed various theories with reference to the genetic mechanism of the ventricular strain pattern ECG.
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  • TAKASI YANAGA
    1961 Volume 25 Issue 12 Pages 1294-1304
    Published: December 15, 1961
    Released on J-STAGE: April 14, 2008
    JOURNAL FREE ACCESS
    The aconitine-induced fibrillation of the right atrium of the young rabbit was studied by the simultaneous recording of the transmembrane potentials of two myocardial cells with intracellular microelectrodes. The results obtained were as follows : 1. After aconitine application, the ordinary spontaneous contraction of the atrium was inhibited, and then atrial fibrillation gradually developed. At the beginning of the fibrillation, coordination and synchronism of the electrical activities between two cells were fairly well maintained with relatively regular rhythm and amplitude of the action potential. Irregularity of the single cell activity, however, increaced with time, though the grade and temporal process of appearance of fibrillation varied with aconitine concentration. 2. When fibrillation was extinguished, regular rhythm and amplitude of action potentials usually recovered. But the amplitude of the action potentials appeared still decaying with time, and irregularities of the potentials were maintained. The action of aconitine on the atrium was therefore not completely reversible. 3. Coordination or synchronism between the electrical activities of the two fibers of the fibrillating atrial muscle was found to depend not only on the direction (transverse or longitudinal) of those fibers, but also on the distance between two microelectrodes. 4. Two types of "slow" or "small" depolarizations were recorded from the fibrillating atrial muscle. The first type was a oscillatory slow potential in the ectopic pace-maker, and the second type was a "junctional potential", which appeared to be produced by an electrotonic spread of the action potential of the adjoining fiber through the intercellular junction. 5. It was discussed that the behaviour of the fibrillating atrial muscle is determined by the degree of decrease of the safety factor of the intercellular junction, in other words by the "junctional potential".
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  • KENZO KUSUI, MIKIO OKAMOTO
    1961 Volume 25 Issue 12 Pages 1305-1307
    Published: December 15, 1961
    Released on J-STAGE: April 14, 2008
    JOURNAL FREE ACCESS
    Authors recorded a clinical details of Adams-Stokes syndrome in 76 aged man, occurring frequently and disappearing with recovery after some 5 days from the onset. Electrocardiogram showed complete auriculoventricular block. During this time, ventricular asystole was observed and the patient was recovered from faintness soon after the reaction of ventricular systole. Judging from his anamnesis, complete auriculoventricular block in this case may be associated with the hypertensive cardiovascular disturbances.
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