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Junji TAKEUCHI
1964 Volume 40 Issue 3 Pages
141-144
Published: 1964
Released on J-STAGE: September 12, 2006
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Hajime NISHIMURA
1964 Volume 40 Issue 3 Pages
145-149
Published: 1964
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Miyoko UCHIYAMA, Saburo UCHIYAMA
1964 Volume 40 Issue 3 Pages
150-154
Published: 1964
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Tosiro TSUZUKU
1964 Volume 40 Issue 3 Pages
155-156
Published: 1964
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Edward J. TULLY
1964 Volume 40 Issue 3 Pages
157-160
Published: 1964
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Mitsuru NAKAI
1964 Volume 40 Issue 3 Pages
161-165
Published: 1964
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Michihiko KAWAMURA
1964 Volume 40 Issue 3 Pages
166-170
Published: 1964
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Yôto KUBOTA
1964 Volume 40 Issue 3 Pages
171-175
Published: 1964
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Akihiro OKUYAMA
1964 Volume 40 Issue 3 Pages
176-179
Published: 1964
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Masahiro NAKAMURA, Midori YOSHIDA
1964 Volume 40 Issue 3 Pages
180-182
Published: 1964
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Miss Mira SEN
1964 Volume 40 Issue 3 Pages
183-187
Published: 1964
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Kazuo ISHIGURO
1964 Volume 40 Issue 3 Pages
188-191
Published: 1964
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Kazuo ISHIGURO
1964 Volume 40 Issue 3 Pages
192-195
Published: 1964
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Teruo IKEGAMI
1964 Volume 40 Issue 3 Pages
196-199
Published: 1964
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Hideo YAMAGATA
1964 Volume 40 Issue 3 Pages
200-205
Published: 1964
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Yasushi NAKAMURA
1964 Volume 40 Issue 3 Pages
206-210
Published: 1964
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The reduction kinetics of the finely powdered iron oxide was investigated in the temperature range from 450° to 550°C. The rate determining step of the reduction of hematite and magnetite by hydrogen was found to be typically topochemical reaction at the interface of oxide and metal. The proposed model for the reduction mechanism was supported by the experimental results.
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Hiroaki NISHIKAWA, Shoichiro YAMADA, Emiko YOSHIDA
1964 Volume 40 Issue 3 Pages
211-213
Published: 1964
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Hisakatsu YABE
1964 Volume 40 Issue 3 Pages
214-219
Published: 1964
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Susumu SHIBATA, Akira TAMURA, Iwao IUCHI, Takaoki MIYAJI
1964 Volume 40 Issue 3 Pages
220-225
Published: 1964
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Hb MIwate is a new abnormal hemoglobin that had never been recorded prior to 1960. It is the cause of cyanosis in the congenital nigremia which is inherited as an autosomal dominant character. The disease may be considered on the basis of the chemical study described above as follows:-The nigremia is a hemoglobinopathy due to a mutation that has caused substitution by tyrosine for the histidine of the 87th amino acid residue. The patient of this disease is the heterozygote to be designated by the genetic formula (αMI/αΛ, βΛ/βΛ): the homozygote (αMI/αMI, βΛ/βΛ) has not yet been discovered. Possibly, this combination will act as a lethal on the fetus on account of deficient oxygenation of the fetal circulation.
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Yoshito YAMASAKI, Nobuo MURATA, Takashi SUWA
1964 Volume 40 Issue 3 Pages
226-231
Published: 1964
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Eiji FUKUSHIMA, Shoichi IWASA
1964 Volume 40 Issue 3 Pages
232-235
Published: 1964
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Sueo TATSUOKA, Kuniyoshi TANAKA, Masuo MIYAMOTO, Katsura MORITA, Yutak ...
1964 Volume 40 Issue 3 Pages
236-240
Published: 1964
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Hiroshi IRISAWA, Naoko NISIMARU
1964 Volume 40 Issue 3 Pages
241-244
Published: 1964
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The delay of conduction of excitation from the ventricle to the bulbus cordis in the frog's heart took place mainly at a narrow junctional area at the ventricular side of the bulbus cordis tissue. The action potential of this tissue showed a slow rate of depolarization which is different from that seen in the bulbus cordis and the ventricle.
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