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Volume 30, Issue 5
Displaying 1-17 of 17 articles from this issue
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[in Japanese]1998 Volume 30 Issue 5 Pages 376
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSDownload PDF (150K) -
Dipole Tracing MethodKimiko Yasuda, Junichi Takanashi, Katsuo Sugita, Hiroo Niimi, Hiroto I ...1998 Volume 30 Issue 5 Pages 377-382
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSWe studied the location of the electric generator of photoparoxysmal discharges using a scalp- skullbrain dipole tracing (DT) method. By this method, the location of epileptic discharges on each scalp EEG was calculated as an equivalent current dipole (ECD) in 8 cases of epilepsy who had photoparoxysmal discharges. We divided these cases into groups A and B (4 cases each), comprising of patients with and without visually-induced seizures, respectively. The ECDs of the 3 cases in group A corresponded to the small area adjacent to the lateral geniculate nucleus on MRI superimposed. On the other hand, the ECDs of all cases in group B located at the corpus callosum. This study suggests the pathways of epileptic discharges from the epileptic focus are different between two groups. It is supposed that neural activity of the lateral geniculate nucleus might be responsible for the generator mechanism of photoparoxysmal discharges which evokes photosensitive epilepsy.View full abstractDownload PDF (6284K) -
Akashi Ishikawa1998 Volume 30 Issue 5 Pages 383-386
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSThe author reported eleven patients with brainstem edema on cranial CT. Nine had acute encephalopathy or Reye syndrome and the other two had acute encephalitis. Seven cases died and four cases survived. Dead cases showed brainstem edema significantly faster than the survivers. Laboratory examinations revealed significant differences of LDH, platelet and HCO3- between the two groups. Six patients died in the winter. The duration of convulsion was more than 30 minutes in all the dead cases.View full abstractDownload PDF (3422K) -
Hideji Hattori, Hiroshi Inada, Katsuji Tanaka, Shizuhiro Niihira, Tosh ...1998 Volume 30 Issue 5 Pages 387-393
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSAuditory brainstem responses (ABR) and other evoked potentials were studied in four patients with the Wolfram syndrome. In three cases ABR was abnormal in the early stage of the disease. There was no responses or only the wave V with prolonged latency at a stimulation level of 80 dBnHL. A prolongation of the I - V interpeak latency (IPL) was also revealed at a stimulation level of 105 dBnHL. The remaining patient showed shortening of the I - V IPL. The visual evoked potentialshowed prolonged peak latency in three cases, and the median nerve short latency somatosensory evoked potentials were normal in two cases. These ABR findings indicated not only sensory neuronal hearing loss but also a degenerative change in the brain stem in the Wolfram syndrome.View full abstractDownload PDF (758K) -
Junji Yamada, Kazuyoshi Morimoto, Osamu Takemoto1998 Volume 30 Issue 5 Pages 394-397
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSA 5- year- old boy with hydrocephalus and a lumbar myelomeningocele underwent extension of peritoneal tube of ventriculoperitoneal (VP) shunt system. Prior to the operation he had been able to walk independently with the use of braces, to speak complex sentences and to sing songs. After the surgery, he lost consciousness and became critically ill with irregular respiration. He was artificially ventilated for 10 days, and then recovered, with sequelae of right facial paresis and slight dysarthria. T1 weighted magnetic resonance image showed high intensities of the medial part of the inferior lobe of the cerebellum and medulla oblongata, which were interpreted as representing edema. This case illustrates that Chiari II malformation can become symptomatic after a VP shunt trouble.View full abstractDownload PDF (3847K) -
Toshio Saito, Yuri Chikumaru, Takeshi Koriyama, Yasushi Yoshioka, Hiro ...1998 Volume 30 Issue 5 Pages 398-402
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSWe experienced a case of acute necrotizing encephalopathy of childhood with a good outcome. The disease continued for about three weeks, and the patient recovered with a sequela of mild left-hemiparesis. On the brain magnetic resonance imaging, abnormal intensity areas in the thalamus and putamen reduced size at an early stage of the disease. After 1 year, his mental and motor development seemed to be almost normal, but brain single photon emission computed tomography and electroencephalography were abnormal. Subclinical functional abnormalities persisted longer than had been expected.View full abstractDownload PDF (6893K) -
Tsukasa Higuchi, Yuji Inaba, Yukiko Hata, Chinatsu Seki, Motoki Ichika ...1998 Volume 30 Issue 5 Pages 403-409
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSMagnetic resonance imaging (MRI), single photon emission computed tomography (SPECT) and magnetic resonance spectroscopy (MRS) were successively recorded in a 3-year-old girl with the acute hemiplegia syndrome. She was admitted to our hospital with complaints of fever, loss of consciousness and right side dominant clonic convulsions evolving into status epilepticus, and then recovered with sequelae of aphasia and right hemiparesis. Electroencephalography showed a generalized slow rhythm at the onset, and very low activities on the left hemisphere in the follow-up records. Brain CT and MRI revealed edema of the left hemisphere initially, followed by left side dominant brain atrophy. No cerebral vascular lesion was detected by magnetic resonance angiography. N-Isopropyl-[123I]-iodoamphetamine SPECT showed marked hypoperfusion of the left hemisphere accompanied by crossed cerebellar diaschisis. MRS at the initial stage detected decreased N-acetyl-aspartic acid and increased lactic acid signals in the bilateral hemisphere, which subsequently normalized only on the right side. These findings suggested brain damage and neural cell death in the left cerebral hemisphere, caused by acute encephalopathy. SPECT and MRS are useful new techniques to study the pathophysiology of the acute hemiplegia syndrome.View full abstractDownload PDF (5623K) -
Yasuhiro Suzuki, Osamu Takemoto, Hiroshi Arai, Megumi Goto, Junji Yama ...1998 Volume 30 Issue 5 Pages 411-416
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSWe report a case of congenital toxoplasmosis with a prenatal diagnosis of fetal hydrocephalus. A CT scan performed at birth revealed ventricular dilatation with calcification. The serum and CSF Toxoplasma specific IgM were elevated. The diagnosis was confirmed by the presence of Toxoplasma cysts in the placenta, and detection of the Toxoplasma SAG1 gene by a polymerase chain reaction (PCR) (placenta, positive; CSF, negative). Signs of active central nervous system infection, such as the decreased CSF glucose, and elevated CSF protein, neuron specific enolase and LDH, resolved after initiation of treatment with pyrimethamine and sulfazoxine. A PCR test using the placental tissue may be useful for the rapid diagnosis of congenital toxoplasmosis.View full abstractDownload PDF (4463K) -
Hideyuki Nagai, Akane Shikata, Noriko Sato, Yoshihiro Takeuchi, Tadash ...1998 Volume 30 Issue 5 Pages 417-422
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSWe report an 11-year- old boy with a non-photosensitive epileptic self-induced seizures, pacygyria and familial ataxia. His grandmother and aunts had dysarthria, and his mother had developed progressive ataxia and myoclonus since 40 years old. His older sister had ataxia, mental retardation and epilepsy. As for the boy, motor developmental delay with muscle hypertonicity of left extremities was recognized at the age of 5 months. Mental retardation and ataxia was recognized at the age of 3 years and slight mental regression is recognized at the age of 11 years. No special findings were detected in an examination of his blood and cerebrospinal fluid, including amino acids, lysos6mal enzymes activity and genetic analysis for dentatorubralpallidoluysian atrophy. Brain magnetic resonance imaging revealed pachygyria of the right cerebral cortecies. At the age of two, he began to induce seizures with impairment of consciousness in himself by waving his right hand over his face which was directed toward a source of bright light. At the age of seven, he developed spontaneous seizures with impairment of consciousness. An EEG showed frequent spikes in the occipital area, on the right and left sides occurring either independently or synchronously. Intermittent photic stimulation and pattern stimulation did not induce a paroxysmal discharge in EEG. Ictal EEG suggested that the origin of the seizures was the occipital lobe. Treatment with valproate and zonisamide was effective in reducing the seizures. The findings of our case imply the pathogenesis of self- induced seizures and the relationship between PME and neuronal migration disorders.View full abstractDownload PDF (3509K) -
Possible Evidence of E ncephalo-Myelo-Radiculo-NeuropathyNaoya Itokazu, Yukiko Kodama, Sachiyo Kontani, Shinobu Inoue, Tohru Su ...1998 Volume 30 Issue 5 Pages 423-429
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSWe report a case of acute polyradiculoneuritis with multiple cranial nerve palsy and cerebral lesions. A boy, born on July 26, 1987, developed unusual sensation on the extremities, backache and sleep disturbance on June 23, 1996. On July 2, following a complaint of blindness he developed a convulsion and was admitted to our hospital. Neurological examination revealed intact consciousness, severe external ophthalmoplegia, bifacial palsy and generalized areflexia. On the next day, flaccid tetraplegia and respiratory dysfunction developed and progressed without disturbance of consciousness. After tracheal intubation he was under mechanical ventilation. A lumbar puncture examination showed clear CSF with increased protein 166 mg/dl, no cells and normal myelin basic protein. Serum antibodies against gangliosides (GM1, GD. and GQ.) were not detected. A posterior tibial nerve conduction velocity was mildly delayed with disappearance of F- wave. On the other hand, very slow background activity was shown by EEG, extensive focal hypoperfusion of cerebral blood flow by SPECT and supratentorial multiple high intensity lesions by T2 weighted MRI of the brain. There were no abnormal signals in the brainstem and cerebellum on MRI. His condition dramatically improved after plasmapheresis. The abnormal findings of SPECT and MRI promptly disappeared within 3 weeks, although abnormal signs on EEG persisted. He was successfully weaned off the respirator and recovered strength of the limbs. He was discharged on August 28, 1996, with supported walk and bifacial palsy, then he completely recoverd by 7 months. The condition of case was compatible with ‘encephalo-myelo-radiculo-neuropathy’, adisease entity that had previously been reported in a few patients in whom with Guillain-Barre or Fisher syndrome and cerebral symptoms co-existed.View full abstractDownload PDF (7179K) -
[in Japanese], [in Japanese], [in Japanese]1998 Volume 30 Issue 5 Pages 431-433
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESS -
[in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...1998 Volume 30 Issue 5 Pages 433-435
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESS -
[in Japanese], [in Japanese], [in Japanese], [in Japanese], [in Japane ...1998 Volume 30 Issue 5 Pages 435-437
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESS -
[in Japanese], [in Japanese]1998 Volume 30 Issue 5 Pages 440
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSDownload PDF (132K) -
[in Japanese]1998 Volume 30 Issue 5 Pages 443-444
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSDownload PDF (121K) -
[in Japanese]1998 Volume 30 Issue 5 Pages 445
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSDownload PDF (157K) -
[in Japanese], [in Japanese]1998 Volume 30 Issue 5 Pages 453-454
Published: September 01, 1998
Released on J-STAGE: August 10, 2011
JOURNAL FREE ACCESSDownload PDF (259K)
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